Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Rose Anna Kostrzewa"'
Publikováno v:
Neurotoxicity Research. 17:299-304
The object of the current study was to determine the effect of L: -3,4-dihydroxyphenylalanine (L: -DOPA) on the in vivo striatal microdialysate levels of the respective dopamine and serotonin metabolites 3,4-dihydroxyphenlalanine (DOPAC) and 5-hydrox
Autor:
John P, Kostrzewa, Rose Anna, Kostrzewa, Richard M, Kostrzewa, Ryszard, Brus, Przemysław, Nowak
Publikováno v:
Current topics in behavioral neurosciences. 29
The classic rodent model of Parkinson's disease (PD) is produced by unilateral lesioning of pars compacta substantia nigra (SNpc) in adult rats, producing unilateral motor deficits which can be assessed by dopamine (DA) D
Autor:
John P, Kostrzewa, Rose Anna, Kostrzewa, Richard M, Kostrzewa, Ryszard, Brus, Przemysław, Nowak
Publikováno v:
Current topics in behavioral neurosciences. 29
The neonatally 6-hydroxydopamine (n6-OHDA)-lesioned rat has been the standard for 40 years, as an animal model of attention-deficit hyperactivity disorder (ADHD). Rats so lesioned during postnatal ontogeny are characterized by ~99 % destruction of do
Publikováno v:
Amino Acids. 28:157-164
L-Dihydroxyphenylalanine (L: -DOPA), the anti-parkinsonian drug affording the greatest symptomatic relief of parkinsonian symptoms, is still misunderstood in terms of its neurotoxic potential and the mechanism by which generated dopamine (DA) is able
Publikováno v:
Neurotoxicity Research. 6:457-462
Receptor priming is a recently discovered phenomenon by which receptor agonists produce abrupt and long-lived supersensitization of receptors. Induction of dopamine (DA) D2 receptor supersensitivity by the agonist quinpirole was discovered approximat
Publikováno v:
Neurotoxin Modeling of Brain Disorders—Life-long Outcomes in Behavioral Teratology ISBN: 9783319341347
The classic rodent model of Parkinson’s disease (PD) is produced by unilateral lesioning of pars compacta substantia nigra (SNpc) in adult rats, producing unilateral motor deficits which can be assessed by dopamine (DA) D2 receptor (D2-R) agonist i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::eb3c97ed622784e9129c977b60ae270d
https://doi.org/10.1007/7854_2015_396
https://doi.org/10.1007/7854_2015_396
Publikováno v:
Neurotoxin Modeling of Brain Disorders—Life-long Outcomes in Behavioral Teratology ISBN: 9783319341347
The neonatally 6-hydroxydopamine (n6-OHDA)-lesioned rat has been the standard for 40 years, as an animal model of attention-deficit hyperactivity disorder (ADHD). Rats so lesioned during postnatal ontogeny are characterized by ~99 % destruction of do
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ece95793b868d592afd9b1b1e9aad758
https://doi.org/10.1007/7854_2015_397
https://doi.org/10.1007/7854_2015_397
Autor:
Richard M. Kostrzewa, John P. Kostrzewa, Rose Anna Kostrzewa, Florence P. Kostrzewa, Ryszard Brus, Przemyslaw Nowak
Publikováno v:
Staging Neuropsychiatric Disorders ISBN: 9781461407843
Staging Neuropsychiatric Disorders ISBN: 9781461472636
Staging Neuropsychiatric Disorders ISBN: 9781461472636
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ee889d40facbe608d2c663cb34fb4921
https://doi.org/10.1007/978-1-4614-0785-0_13
https://doi.org/10.1007/978-1-4614-0785-0_13
Autor:
Rose Anna Kostrzewa, Florence P. Kostrzewa, Richard M. Kostrzewa, Ryszard Brus, John P. Kostrzewa, Przemysław Nowak
Publikováno v:
Neurotoxicity research. 19(2)
Dopamine receptor supersensitivity (DARSS) often is invoked as a mechanism possibly underlying disordered thought processes and agitation states in psychiatric disorders. This review is focused on identified means for producing DARSS and associating
Publikováno v:
Journal of neural transmission (Vienna, Austria : 1996). 115(2)
For more than 50 years, heavy metal exposure during pre- or post-natal ontogeny has been known to produce long-lived hyperactivity in rodents. Global brain injury produced by neonatal hypoxia also produced hyperactivity, as did (mainly) hippocampal i