Zobrazeno 1 - 10
of 27
pro vyhledávání: '"Rong‐Quan Fu"'
Publikováno v:
Cellular Physiology and Biochemistry, Vol 51, Iss 5, Pp 2111-2122 (2018)
Background/Aims: Sestrin 2 is associated with the pathophysiology of several diseases. The aim of this study was to investigate the effects and potential mechanisms of Sestrin 2 in rat hepatic stellate cells (HSCs) during liver fibrogenesis. Methods:
Externí odkaz:
https://doaj.org/article/5846f4a42a944f87abd5b03a47cc4e30
Publikováno v:
Gastroenterology Research and Practice, Vol 2018 (2018)
Background. Type 2 diabetes mellitus (T2DM) patients are involved closely with cancer. This work aims to conduct a systematic review and network meta-analysis (NMA) to examine the effect of different types of statins on cancer incidence in patients w
Externí odkaz:
https://doaj.org/article/28a829aec1b84959a3d730dbcf1399cb
Autor:
Xiao‐Dong Wang, Chen‐Wei Pan, Guang‐Yao Zhou, Feng Gao, Feng‐Ling Wang, Rong‐Quan Fu, Er‐Hui Xiao, Peng Li, Huai Zhang, Ming‐Hua Zheng
Publikováno v:
Journal of viral hepatitisREFERENCES. 29(3)
Liver steatosis is becoming increasingly common in patients with chronic hepatitis B (CHB), and its effect on liver stiffness measurement (LSM), as assessed by transient elastography, remains controversial. Seven hundred and fifty-five patients with
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a85676e67fef893715ce9c351a8ab6e7
https://pubmed.ncbi.nlm.nih.gov/34902216
https://pubmed.ncbi.nlm.nih.gov/34902216
Autor:
RONG‑QUAN FU1, DAN‑PING HU1, YI‑BING HU1, LIANG HONG1, QING‑FENG SUN1, JI‑GUANG DING1 djg5011@163.com
Publikováno v:
Molecular Medicine Reports. Oct2017, Vol. 16 Issue 4, p4327-4333. 7p.
Publikováno v:
Molecular Medicine Reports.
Hypoxia serves a critical role in the pathogenesis of liver fibrosis. Hypoxia‑inducible factor 1α (HIF1‑α) is induced when cells are exposed to low O2 concentrations. Recently, it has been suggested that Rho‑associated coiled‑coil‑forming
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9851cdd94ab2444fd5dfc7bbb29b69af
https://doi.org/10.3892/mmr.2018.9397
https://doi.org/10.3892/mmr.2018.9397
Publikováno v:
Molecular Medicine Reports. 13:224-230
Liver fibrosis is a wound-healing response to chronic liver injury that results in the accumulation of extracellular matrix proteins. It eventually leads to cirrhosis of the liver and liver failure, and it is a critical threat to the health and lives
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f2ac946e9eaa381d402f67e32361b89b
https://doi.org/10.3892/mmr.2015.4497
https://doi.org/10.3892/mmr.2015.4497
Publikováno v:
Minerva medica. 108(3)
INTRODUCTION The aim of this study was to evaluate the correlation of High-mobility group box 1 (HMGB1) expression in the serum with chronic hepatitis B (CHB) related liver fibrosis, severe hepatitis B and acute-on-chronic liver failure (ACLF). EVIDE
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::988d8ccd74740c5c23f17c40ee823b44
https://pubmed.ncbi.nlm.nih.gov/27973467
https://pubmed.ncbi.nlm.nih.gov/27973467
Publikováno v:
Journal of hepatology. 66(1)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a8be718e7185bdf9722855dfceae8467
https://pubmed.ncbi.nlm.nih.gov/27743988
https://pubmed.ncbi.nlm.nih.gov/27743988
Publikováno v:
Alimentary pharmacologytherapeutics. 44(7)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::83849f5c6fc7784365c1ba46b5724d70
https://pubmed.ncbi.nlm.nih.gov/27593427
https://pubmed.ncbi.nlm.nih.gov/27593427
Publikováno v:
Molecular medicine reports. 16(4)
The aim of the present study was to investigate the role of microRNA (miR)‑21 in regulating collagen I and Smad7 expression in activated rat hepatic stellate cells (HSCs). Rat HSCs were isolated by single‑step density gradient centrifugation with
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4053ccdd521cbd86074afbd2ad78e60b
https://pubmed.ncbi.nlm.nih.gov/28731181
https://pubmed.ncbi.nlm.nih.gov/28731181