Zobrazeno 1 - 10
of 220
pro vyhledávání: '"Ronald R. Magness"'
Autor:
Shannon J. Ho, Dale Chaput, Rachel G. Sinkey, Amanda H. Garces, Erika P. New, Maja Okuka, Peng Sang, Sefa Arlier, Nihan Semerci, Thora S. Steffensen, Thomas J. Rutherford, Angel E. Alsina, Jianfeng Cai, Matthew L. Anderson, Ronald R. Magness, Vladimir N. Uversky, Derek A. T. Cummings, John C. M. Tsibris
Publikováno v:
Cell Communication and Signaling, Vol 22, Iss 1, Pp 1-23 (2024)
Abstract VEGFR2 (Vascular endothelial growth factor receptor 2) is a central regulator of placental angiogenesis. The study of the VEGFR2 proteome of chorionic villi at term revealed its partners MDMX (Double minute 4 protein) and PICALM (Phosphatidy
Externí odkaz:
https://doaj.org/article/4a1dc85d29c94f1b8404ba416c726589
Autor:
Nihan Günay, Ronald R. Magness, Charles J. Lockwood, Anthony N. Imudia, Umit A. Kayisli, Frederick Schatz, Ozlem Guzeloglu-Kayisli, Esma Kirimlioglu, Rachel Grimes Sprague, Asli Ozmen, Xiaofang Guo, Joung Woul Kim
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 106:337-350
Context Enhanced levels of catecholestradiols, 2-hydroxyestradiol (2-OHE2) or 4-hydroxyestradiol (4-OHE2), are reported in endometriosis. During gestation, catecholestradiol activation of adrenergic receptors (AR) elevates estrogen receptor (ER)-inde
Normal pregnancy is associated with dramatic increases in uterine blood flow to facilitate the bidirectional maternal–fetal exchanges of respiratory gases and to provide sole nutrient support for fetal growth and survival. The mechanism(s) underlyi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::725131962201459aa4ce62fed462b2a0
https://doi.org/10.9734/bpi/cacb/v8/9373d
https://doi.org/10.9734/bpi/cacb/v8/9373d
Autor:
Kevin Cowart, Ronald R. Magness, Nicholas W. Carris, Srinivas M. Tipparaju, Athanasios Tsalatsanis, Ambuj Kumar, Byron Cheon
Publikováno v:
J Gen Intern Med
Autor:
Qing-Yun Zou, Chi Zhou, Ronald R. Magness, Xin-Qi Zhong, Qin Yan, Chanel T. Tyler, Jing Zheng, Ian M. Bird
Publikováno v:
Hypertension
Preeclampsia impairs fetoplacental vascular function and increases risks of adult-onset cardiovascular disorders in children born to preeclamptic mothers, implicating that preeclampsia programs fetal vasculature in utero. However, the underlying mech
Publikováno v:
Reproductive Sciences. 26:459-468
Nitric oxide (NO) production is essential to facilitate rises in uterine blood flow (UBF) during pregnancy. It has been proposed that the metabolites of E(2)β, 2-hydroxyestradiol (2-OHE(2)), 4-hydroxyestradiol (4-OHE(2)), 2-methoxyestradiol (2-ME(2)
Publikováno v:
Eicosanoids in Reproduction ISBN: 9781315150949
Eicosanoids in Reproduction
Eicosanoids in Reproduction
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::1dd434c9eb187187a6faaecd07b8708c
https://doi.org/10.1201/9781315150949-9
https://doi.org/10.1201/9781315150949-9
Autor:
Kevin M. Johnson, Ronald R. Magness, Philip A. Corrado, Thaddeus G. Golos, Oliver Wieben, Christopher J. François, Dinesh Shah, Jacob A. Macdonald, Sydney M. Nguyen
Publikováno v:
Journal of Magnetic Resonance Imaging. 49:534-545
Background Pregnancy complications are often associated with poor uteroplacental vascular adaptation and standard diagnostics are unable to reliably quantify flow in all uteroplacental vessels and have poor sensitivity early in gestation. Purpose To
Autor:
Ronald R. Magness, Feng Cheng, Nicholas W. Carris, Athanasios Tsalatsanis, Ambuj Kumar, Srinivas M. Tipparaju
Publikováno v:
Diabetes, Obesity and Metabolism. 20:1994-1999
Statins are widely prescribed, yet statin muscle pain limits their use, leading to increased cardiovascular risk. No validated therapy for statin muscle pain exists. The goal of the study was to assess whether metformin was associated with reduced mu
Publikováno v:
Hypertension (Dallas, Tex. : 1979)
Supplemental Digital Content is available in the text.
Cell–cell communication is dependent on GJ (gap junction) proteins such as Cx43 (connexin 43). We previously demonstrated the importance of Cx43 function in establishing the enhanced pregn
Cell–cell communication is dependent on GJ (gap junction) proteins such as Cx43 (connexin 43). We previously demonstrated the importance of Cx43 function in establishing the enhanced pregn