Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Rohit Nalavade"'
Autor:
Kristoffer Unterbruner, Frank Matthes, Judith Schilling, Rohit Nalavade, Stephanie Weber, Jennifer Winter, Sybille Krauß
Publikováno v:
PLoS ONE, Vol 13, Iss 1, p e0190437 (2018)
The MID1 ubiquitin ligase activates mTOR signaling and regulates mRNA translation. Misregulation of MID1 expression is associated with various diseases including midline malformation syndromes, cancer and neurodegenerative diseases. While this indica
Externí odkaz:
https://doaj.org/article/f14f8b1465b44cfe9d2601e0213de085
Autor:
Bernd O Evert, Rohit Nalavade, Johannes Jungverdorben, Frank Matthes, Stephanie Weber, Ashish Rajput, Stefan Bonn, Oliver Brüstle, Michael Peitz, Sybille Krauß
Publikováno v:
PLoS ONE, Vol 13, Iss 8, p e0201794 (2018)
Molecular chaperones are important regulators of protein folding and proteasomal removal of misfolded proteins. In spinocerebellar ataxia type 3 (SCA3), the co-chaperone DnaJ homology subfamily B member 1 (DNAJB1 or heat shock protein 40) is recruite
Externí odkaz:
https://doaj.org/article/536afac7b61e4d8e8821a7217b4f1e15
Publikováno v:
MicroRNA 8(1), 76-85 (2018). doi:10.2174/2211536607666180821162403
Background: Spinocerebellar ataxia type 3 (SCA3), the most common spinocerebellar ataxia, is caused by a polyglutamine (polyQ) expansion in the protein ataxin-3 (ATXN3). Silencing the expression of polyQ-expanded ATXN3 rescues the cellular disease ph
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2d5ecaf4ba17989f8e6a01f4496a4f01
https://pubmed.ncbi.nlm.nih.gov/30147021
https://pubmed.ncbi.nlm.nih.gov/30147021
Autor:
Sybille Krauß, Kristoffer Unterbruner, Judith Schilling, Frank Matthes, Rohit Nalavade, Jennifer Winter, Stephanie Weber
Publikováno v:
PLOS ONE 13(1), e0190437 (2018). doi:10.1371/journal.pone.0190437
PLoS ONE, Vol 13, Iss 1, p e0190437 (2018)
PLoS ONE
PLoS ONE, Vol 13, Iss 1, p e0190437 (2018)
PLoS ONE
The MID1 ubiquitin ligase activates mTOR signaling and regulates mRNA translation. Misregulation of MID1 expression is associated with various diseases including midline malformation syndromes, cancer and neurodegenerative diseases. While this indica
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::016659325540e2588fbb48cd7a64c806
Autor:
Ireen König, Stephanie Weber, Christoph Möhl, Sybille Krau, Verena Pesch, Rohit Nalavade, Manuel Schölling, Nadine Griesche
Publikováno v:
Journal of Analytical & Bioanalytical Techniques.
Traditionally, studies on protein translation rely on systems, in which cells have been lysed prior determination of levels of the protein of interest. However, these assays do not reflect the protein synthesis in living cells in real time, but analy
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d317a381121cd853c0db9e5f443f7df6
https://doi.org/10.4172/2155-9872.1000390
https://doi.org/10.4172/2155-9872.1000390
Autor:
Sybille Krauß, Frank Matthes, Bernd O. Evert, Oliver Brüstle, Johannes Jungverdorben, Ashish Rajput, Michael Peitz, Stephanie Weber, Rohit Nalavade, Stefan Bonn
Publikováno v:
PLoS ONE
PLoS ONE, Vol 13, Iss 8, p e0201794 (2018)
PLOS ONE 13(8), e0201794 (2018). doi:10.1371/journal.pone.0201794
PLoS ONE, Vol 13, Iss 8, p e0201794 (2018)
PLOS ONE 13(8), e0201794 (2018). doi:10.1371/journal.pone.0201794
Molecular chaperones are important regulators of protein folding and proteasomal removal of misfolded proteins. In spinocerebellar ataxia type 3 (SCA3), the co-chaperone DnaJ homology subfamily B member 1 (DNAJB1 or heat shock protein 40) is recruite
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d73056e927965b7cc6a8a03e0f5c25c7
https://doi.org/10.1371/journal.pone.0201794
https://doi.org/10.1371/journal.pone.0201794
Publikováno v:
Cell Death & Disease
Cell death & disease 4(8), e752-e752 (2013). doi:10.1038/cddis.2013.276
Cell death & disease 4(8), e752-e752 (2013). doi:10.1038/cddis.2013.276
Several inherited neurodegenerative disorders are caused by CAG trinucleotide repeat expansions, which can be located either in the coding region or in the untranslated region (UTR) of the respective genes. Polyglutamine diseases (polyQ diseases) are
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::376ecb9880579744c561932ddb034e0e
https://pubmed.ncbi.nlm.nih.gov/23907466
https://pubmed.ncbi.nlm.nih.gov/23907466