Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Rodney J Morel"'
Autor:
Anke M Schulte, Seng H. Cheng, Cliff Kremer, Kristin M. Taylor, Birgit Meyer-Puttlitz, Ronald K. Scheule, Jeffrey Bajko, Rodney J Morel, Mario S Cabrera
Publikováno v:
Journal of Diabetes & Metabolism.
The mammalian target of rapamycin complex 1 (mTORC1) regulates insulin-mediated glucose metabolism, cell proliferation, the oxidative branch of the pentose phosphate pathway, de novo lipogenesis, and autophagy. Ribosomal S6 kinase 1 (S6K1) and 2 (S6K
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::eb0ce84bd08b17b6aa5601ef0b6e7015
https://doi.org/10.4172/2155-6156.1000441
https://doi.org/10.4172/2155-6156.1000441
Autor:
Nicholas P Clayton, Carol A Nelson, Timothy Weeden, Kristin M Taylor, Rodney J Moreland, Ronald K Scheule, Lucy Phillips, Andrew J Leger, Seng H Cheng, Bruce M Wentworth
Publikováno v:
Molecular Therapy: Nucleic Acids, Vol 3, Iss C (2014)
Pompe disease is an autosomal recessive disorder caused by a deficiency of acid α-glucosidase (GAA; EC 3.2.1.20) and the resultant progressive lysosomal accumulation of glycogen in skeletal and cardiac muscles. Enzyme replacement therapy using recom
Externí odkaz:
https://doaj.org/article/cc5392267a07419090c8a0e91d1a8ae4
Autor:
Kristin M Taylor, Elizabeth Meyers, Michael Phipps, Priya S Kishnani, Seng H Cheng, Ronald K Scheule, Rodney J Moreland
Publikováno v:
PLoS ONE, Vol 8, Iss 2, p e56181 (2013)
Pompe disease, also known as glycogen storage disease (GSD) type II, is caused by deficiency of lysosomal acid α-glucosidase (GAA). The resulting glycogen accumulation causes a spectrum of disease severity ranging from a rapidly progressive course t
Externí odkaz:
https://doaj.org/article/eb859b5cc58943a8b5ff18a9d84331be