Anti-Ferroptotic Treatment Deteriorates Myocardial Infarction by Inhibiting Angiogenesis and Altering Immune Response.

Autor: Stairley RA; Department of Regenerative Medicine and Cell Biology, Medical University of South Carolina, Charleston, SC 29425, USA., Trouten AM; Department of Regenerative Medicine and Cell Biology, Medical University of South Carolina, Charleston, SC 29425, USA., Li S; Department of Regenerative Medicine and Cell Biology, Medical University of South Carolina, Charleston, SC 29425, USA.; Department of Pediatrics, Herman B Wells Center for Pediatric Research, Indiana University School of Medicine, Indianapolis, IN 46202, USA., Roddy PL; Department of Regenerative Medicine and Cell Biology, Medical University of South Carolina, Charleston, SC 29425, USA., DeLeon-Pennell KY; Division of Cardiology, Department of Medicine, Medical University of South Carolina, Charleston, SC 29425, USA.; Research Service, Ralph H. Johnson Veterans Affairs Medical Center, Charleston, SC 29401, USA., Lee KH; Department of Medicine Digestive Disease Research Core Center, Medical University of South Carolina, Charleston, SC 29425, USA., Sucov HM; Department of Regenerative Medicine and Cell Biology, Medical University of South Carolina, Charleston, SC 29425, USA.; Division of Cardiology, Department of Medicine, Medical University of South Carolina, Charleston, SC 29425, USA., Liu C; Department of Physiology, Medical College of Wisconsin, Milwaukee, WI 53226, USA., Tao G; Department of Regenerative Medicine and Cell Biology, Medical University of South Carolina, Charleston, SC 29425, USA.

Publikováno v: Antioxidants (Basel, Switzerland) [Antioxidants (Basel)] 2024 Jun 26; Vol. 13 (7). Date of Electronic Publication: 2024 Jun 26.

Cardiomyocyte-fibroblast interaction regulates ferroptosis and fibrosis after myocardial injury.

Autor: Mohr ME; Department of Regenerative Medicine and Cell Biology, Medical University of South Carolina, Charleston, SC 29425, USA.; These authors contributed equally., Li S; Department of Regenerative Medicine and Cell Biology, Medical University of South Carolina, Charleston, SC 29425, USA.; These authors contributed equally., Trouten AM; Department of Regenerative Medicine and Cell Biology, Medical University of South Carolina, Charleston, SC 29425, USA., Stairley RA; Department of Regenerative Medicine and Cell Biology, Medical University of South Carolina, Charleston, SC 29425, USA., Roddy PL; Department of Regenerative Medicine and Cell Biology, Medical University of South Carolina, Charleston, SC 29425, USA., Liu C; Stanford Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA 94305, USA.; Department of Medicine, Stanford University School of Medicine, Stanford, CA 94305, USA., Zhang M; Pediatric Translational Medicine Institute, Shanghai Children's Medical Center, Shanghai Jiao Tong University School of Medicine, 200127 Shanghai, China., Sucov HM; Department of Regenerative Medicine and Cell Biology, Medical University of South Carolina, Charleston, SC 29425, USA.; Department of Medicine, Division of Cardiology, Medical University of South Carolina, Charleston, SC 29425, USA., Tao G; Department of Regenerative Medicine and Cell Biology, Medical University of South Carolina, Charleston, SC 29425, USA.

Publikováno v: BioRxiv : the preprint server for biology [bioRxiv] 2023 Feb 08. Date of Electronic Publication: 2023 Feb 08.

Neutral sphingomyelinase 2 (nSMase2) is the primary neutral sphingomyelinase isoform activated by tumour necrosis factor-α in MCF-7 cells.

Autor: Clarke CJ; Department of Biochemistry and Molecular Biology, Medical University of South Carolina, Charleston, SC 29425, USA., Cloessner EA, Roddy PL, Hannun YA

Publikováno v: The Biochemical journal [Biochem J] 2011 Apr 15; Vol. 435 (2), pp. 381-90.