Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Robin Schmidt-Haupt"'
Autor:
Andrea Tannapfel, Ulf Panzer, Christian Krebs, Rolf A.K. Stahl, Robin Schmidt-Haupt, Joachim Velden, Ulrich Wenzel, Jan-Eric Turner, Christoph Fraune, Michael N. Quang
Publikováno v:
American Journal of Hypertension. 25:479-486
BACKGROUND CCR5 is expressed on infiltrating T cells in hypertensive mice and CCR5 antagonists reduce hypertension making CCR5 an interesting target in treatment of hypertension. METHODS To evaluate the role of CCR5 in hypertensive renal and cardiac
Autor:
Dimitrios Tsikas, Karsten Sydow, Jan T. Kielstein, Michaela Pekarova, Crystal A. Gadegbeku, Lukáš Kubala, Stefan Blankenberg, Renke Maas, Louis G. D'Alecy, Robin Schmidt-Haupt, Stephan Willems, Natalie Lund, Jessica L. Slocum, Denise Lau, Rainer H. Böger, Olaf J C Hellwinkel, Dorothee Atzler, Anna Klinke, Edzard Schwedhelm, Eike Christin Von Leitner, Thomas Meinertz, Stephan Baldus, Heimo Ehmke
Publikováno v:
Circulation. 124:2735-2745
Background— The nitric oxide synthase inhibitor asymmetrical dimethylarginine (ADMA) and the leukocyte-derived hemoprotein myeloperoxidase (MPO) are associated with cardiovascular diseases. Activation of monocytes and polymorphonuclear neutrophils
Autor:
Christiane Rüster, Christoph Fraune, Christian Krebs, Sybille Franke, Gunter Wolf, Ulrich Wenzel, Robin Schmidt-Haupt
Publikováno v:
American Journal of Nephrology. 34:309-317
Background/Aims: The interaction of ‘advanced glycation end products’ (AGEs) and their receptor ‘RAGE’ plays an important role in diabetic nephropathy. We have previously found that in cultured differentiated podocytes, angiotensin II (ANG II
Autor:
Oliver M. Steinmetz, Heimo Ehmke, Robin Schmidt-Haupt, Rolf A.K. Stahl, Andreas Heilmann, Gabriele Cieslar, Udo Helmchen, Munif Haddad, Edzard Schwedhelm, Birgit Hirsch-Hoffmann, Catherine Meyer-Schwesinger, Ralf A. Benndorf, Rainer H. Böger, Ulrich Wenzel, Friedrich Thaiss, Christian Krebs, Susanne Fehr, Lutz Hein
Publikováno v:
Kidney International. 75(10):1039-1049
Angiotensin II (Ang II) activates at least two receptors, AT1 and AT2, with the majority of its effects-such as vasoconstriction, inflammation, and matrix deposition-mediated by the AT1 receptor. It is thought that the AT2 receptor counteracts these