Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Roberta Altobelli"'
Autor:
Valentina Fodale, Roberta Pintauro, Manuel Daldin, Roberta Altobelli, Maria Carolina Spiezia, Monica Bisbocci, Douglas Macdonald, Alberto Bresciani
Publikováno v:
Scientific Reports, Vol 10, Iss 1, Pp 1-13 (2020)
Abstract Huntington’s disease (HD) is a monogenetic neurodegenerative disorder that is caused by the expansion of a polyglutamine region within the huntingtin (HTT) protein, but there is still an incomplete understanding of the molecular mechanisms
Externí odkaz:
https://doaj.org/article/f0f9716e3bb54bde98e7f84e35b5a6f0
Autor:
Alberto Bresciani, Maria Carolina Spiezia, Roberto Boggio, Cristina Cariulo, Anja Nordheim, Roberta Altobelli, Kirsten Kuhlbrodt, Celia Dominguez, Ignacio Munoz-Sanjuan, John Wityak, Valentina Fodale, Deanna M Marchionini, Andreas Weiss
Publikováno v:
PLoS ONE, Vol 13, Iss 3, p e0194423 (2018)
Autophagy is a cellular mechanism that can generate energy for cells or clear misfolded or aggregated proteins, and upregulating this process has been proposed as a therapeutic approach for neurodegenerative diseases. Here we describe a novel set of
Externí odkaz:
https://doaj.org/article/616beb1cabb749c9965d10a4c5518952
Autor:
Sophie Broadway-Stringer, Camilla Siciliano, Colin E. Murdoch, Milda Bartkeviciute, Roberta Altobelli, Elena De Falco
Publikováno v:
Recent Patents on Regenerative Medicine. 4:149-167
Myocardial infarction results in loss of cardiac muscle and deficiency in cardiac performance. Likewise, peripheral artery disease can result in critical limb ischemia leading to reduced mobility, non-healing ulcers, gangrene and amputation. Both of
Autor:
Cristina Cariulo, Kirsten Kuhlbrodt, Alberto Bresciani, Ignacio Munoz-Sanjuan, Anja Nordheim, Roberto Boggio, Celia Dominguez, Deanna Marchionini, Roberta Altobelli, Maria Carolina Spiezia, John Wityak, Valentina Fodale, Andreas Weiss
Publikováno v:
PLoS ONE, Vol 13, Iss 3, p e0194423 (2018)
PLoS ONE
PLoS ONE
Autophagy is a cellular mechanism that can generate energy for cells or clear misfolded or aggregated proteins, and upregulating this process has been proposed as a therapeutic approach for neurodegenerative diseases. Here we describe a novel set of