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of 3
pro vyhledávání: '"Robert Teachenor"'
Autor:
Denise P. Muñoz, Judith Campisi, Jean-Philippe Coppe, Robert Teachenor, Victoria Chu, Albert R. Davalos, Francis Rodier, Eric Campeau, Dipa Bhaumik, Sahn Ho Kim, Christian Beauséjour, Oanh Le
Publikováno v:
Journal of Cell Science. 124:68-81
DNA damage can induce a tumor suppressive response termed cellular senescence. Damaged senescent cells permanently arrest growth, secrete inflammatory cytokines and other proteins and harbor persistent nuclear foci that contain DNA damage response (D
Autor:
Zhouxin Shen, Robert Teachenor, Cornelis Murre, Lilyan Y.T. Wright, Kristina Beck, Stephen A. Briggs
Numerous in vitro as well as genetic studies have demonstrated that the activities of the E2A proteins are regulated at multiple levels, including modulation of DNA binding by the Id proteins, association with the transcriptional modulators p300 and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3e9d8268ca40b2d3a5c904a6e68870dd
https://europepmc.org/articles/PMC3347234/
https://europepmc.org/articles/PMC3347234/
Autor:
Judith Campisi, Guillaume B Cardin, Francis Rodier, Marco Demaria, David Adler, Pierre-Yves Desprez, Remi-Martin Laberge, Mechtouf N, Robert Teachenor
Publikováno v:
Cell Death & Disease
Senescence is a cellular response to damage and stress. The senescence response prevents cancer by suppressing the proliferation of cells with a compromised genome and contributes to optimal wound healing in normal tissues. Persistent senescent cells