Zobrazeno 1 - 10
of 41
pro vyhledávání: '"Robert P. Bennett"'
Autor:
Pei Xiong, Zhihang Xu, Tai-Sing Wu, Tong Yang, Qiong Lei, Jiangtong Li, Guangchao Li, Ming Yang, Yun-Liang Soo, Robert David Bennett, Shu Ping Lau, Shik Chi Edman Tsang, Ye Zhu, Molly Meng-Jung Li
Publikováno v:
Nature Communications, Vol 15, Iss 1, Pp 1-9 (2024)
Abstract Designing high-performance thermal catalysts with stable catalytic sites is an important challenge. Conventional wisdom holds that strong metal-support interactions can benefit the catalyst performance, but there is a knowledge gap in genera
Externí odkaz:
https://doaj.org/article/99cc9dcbd6ef401b84791e60f835b229
Publikováno v:
BioTechniques, Vol 24, Iss 3, Pp 478-482 (1998)
Green fluorescent protein (GFP) and the Zeocin™-resistance gene Sh ble (ZeoR) were fused together to generate a bifunctional protein for the identification and selection of transfected mammalian cells. Expression of this hybrid selectable marker, G
Externí odkaz:
https://doaj.org/article/071ebf35373e4aeb95d794844899a17e
Autor:
Jeanette M. Rotchell, Lauren C. Jenner, Emma Chapman, Robert T. Bennett, Israel Olapeju Bolanle, Mahmoud Loubani, Laura Sadofsky, Timothy M. Palmer
Publikováno v:
PLoS ONE, Vol 18, Iss 2 (2023)
Microplastics (MPs) are ubiquitous in the environment, in the human food chain, and have been recently detected in blood and lung tissues. To undertake a pilot analysis of MP contamination in human vein tissue samples with respect to their presence (
Externí odkaz:
https://doaj.org/article/aed8d2172b3148139d636c0339c96f29
Publikováno v:
Biology, Vol 12, Iss 10, p 1287 (2023)
Peroxisome-proliferator-activated receptor gamma (PPARγ) is a transcription factor with adipogenic, insulin-sensitizing, and antifibrotic properties. Strong PPARγ activators, such as the thiazolidinediones, can induce unwanted effects such as edema
Externí odkaz:
https://doaj.org/article/159a9c7184624b99aec01ba011d624c4
Autor:
Chuhan Zhang, Yu Hang, Weimin Tang, Diptesh Sil, Heather C. Jensen-Smith, Robert G. Bennett, Benita L. McVicker, David Oupický
Publikováno v:
Pharmaceutics, Vol 14, Iss 3, p 669 (2022)
Alcohol-associated liver disease (AALD) is a major cause of liver disorders worldwide. Current treatment options are limited, especially for AALD-associated fibrosis. Promising approaches include RNA interference for miR-155 overexpression in Kupffer
Externí odkaz:
https://doaj.org/article/372bcf1685d943d29308743cd40e5f69
Autor:
Alexandra B. Ysasi, Robert D. Bennett, Willi Wagner, Cristian D. Valenzuela, Andrew B. Servais, Akira Tsuda, Saumyadipta Pyne, Shuqiang Li, Jonna Grimsby, Prapti Pokharel, Kenneth J. Livak, Maximilian Ackermann, Paul C. Blainey, Steven J. Mentzer
Publikováno v:
Frontiers in Medicine, Vol 7 (2020)
Lung regeneration occurs in a variety of adult mammals after surgical removal of one lung (pneumonectomy). Previous studies of murine post-pneumonectomy lung growth have identified regenerative “hotspots” in subpleural alveolar ducts; however, th
Externí odkaz:
https://doaj.org/article/1d3219b82a0046cf97d080489744ae17
Autor:
Robert G. Bennett
Publikováno v:
European Medical Journal Hepatology, Vol 6, Iss 1, Pp 80-87 (2018)
Hepatic fibrosis is a progressive disease with few treatment options outside of transplantation. Relaxin is a member of the insulin/relaxin superfamily of peptide hormones. Originally known for its roles in pregnancy, relaxin promotes reproductive ti
Externí odkaz:
https://doaj.org/article/da114768e5db4e548fba2bc624d74790
Publikováno v:
Biology, Vol 9, Iss 7, p 151 (2020)
Hepatic fibrosis is the accumulation of excess collagen as a result of chronic liver injury. If left unabated, hepatic fibrosis can lead to the disruption of the liver architecture, portal hypertension, and increased risk of progression to cirrhosis
Externí odkaz:
https://doaj.org/article/d183649c25ca442e993e97cc658ea80a
Autor:
Benita L. McVicker, Robert G. Bennett
Publikováno v:
Frontiers in Pharmacology, Vol 8 (2017)
Fibrosis is a major player in cardiovascular disease, both as a contributor to the development of disease, as well as a post-injury response that drives progression. Despite the identification of many mechanisms responsible for cardiovascular fibrosi
Externí odkaz:
https://doaj.org/article/3bfa2c63423c4b02ad8ff33cf3630e82
Autor:
Alexandra B Ysasi, Willi L Wagner, Cristian D Valenzuela, Arne Kienzle, Andrew B Servais, Robert D Bennett, Akira Tsuda, Maximilian Ackermann, Steven J Mentzer
Publikováno v:
PLoS ONE, Vol 12, Iss 5, p e0177921 (2017)
In many mammals, including rodents and humans, removal of one lung results in the compensatory growth of the remaining lung; however, the mechanism of compensatory lung growth is unknown. Here, we investigated the changes in morphology and phenotype
Externí odkaz:
https://doaj.org/article/6a89162ff38343718113254434d57e3d