Zobrazeno 1 - 9 of 9 pro vyhledávání: '"Robert E. Oliver Iii"'
1
Glutaminolysis and glycolysis regulation by troglitazone in breast cancer cells: Relationship to mitochondrial membrane potential
Autor: Tomas Welbourne, Robert E. Oliver Iii, Francesco Turturro, Ellen Friday
Publikováno v: Journal of Cellular Physiology. 226:511-519
We studied the roles of glycolysis and glutaminolysis following an acute reduction in mitochondrial membrane potential (Ψm) induced by the thiazolidinedione troglitazone (TRO) and compared the responses with CCCP-induced depolarization in breast can
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7d10d9d746a49a7b9606bfe7b0c8cd3b
https://doi.org/10.1002/jcp.22360
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2
Troglitazone Regulates Anaplerosis via a Pull/Push Affect on Glutamate Dehydrogenase Mediated Glutamate Deamination in Kidney-derived Epithelial Cells; Implications for the Warburg Effect
Autor: Ellen Friday, Robert E. Oliver Iii, Francesco Turturro, Tomas Welbourne
Publikováno v: Cellular Physiology and Biochemistry. 26:619-628
Mitochondrial Krebs cycle keto acid pool depends upon input from pyruvate and glutamate to maintain homeostasis. We studied the affect of glucose-derived pyruvate removal on compensatory input from glutamine-derived glutamate by accelerated glutamate
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5d94ae3b3e1ee0bdea33a0cccf280f81
https://doi.org/10.1159/000322329
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3
Role of Epidermal Growth Factor Receptor (EGFR)-Signaling Versus Cellular Acidosis Via Na+/H+ Exchanger1(NHE1)-Inhibition in Troglitazone-Induced Growth Arrest of Breast Cancer-Derived Cells MCF-7
Autor: Ellen Friday, Robert E. Oliver Iii, Francesco Turturro, Tomas Welbourne
Publikováno v: Cellular Physiology and Biochemistry. 20:751-762
Purpose:We previously showed that troglitazone (TRO) induces a profound cellular acidosis in MCF-7 cells as a result of inhibiting Na+/H+ exchanger (NHE)1-mediated acid extrusion
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::f921b8dd58288501fc29d79d29b8ba0f
https://doi.org/10.1159/000110435
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8
Troglitazone and pioglitazone interactions via PPAR-gamma-independent and -dependent pathways in regulating physiological responses in renal tubule-derived cell lines
Autor: Itzhak Nissim, Ellen Friday, Robert E. Oliver Iii, Francesco Turturro, Tomas Welbourne
Publikováno v: American journal of physiology. Cell physiology. 292(3)
Troglitazone (Tro) and pioglitazone (Pio) activation of peroxisome proliferator-activated receptor (PPAR)-gamma and PPAR-gamma-independent pathways was studied in cell lines derived from porcine renal tubules. PPAR-gamma-dependent activation of PPAR
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cf9d2121fcfba36966169f067db568e1
https://pubmed.ncbi.nlm.nih.gov/17065204
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9
Troglitazone's rapid and sustained activation of ERK1/2 induces cellular acidosis in LLC-PK1-F+ cells: physiological responses
Autor: Ellen Friday, Robert E. Oliver Iii, Francesco Turturro, Tomas Welbourne, Ashley Lacy
Publikováno v: American journal of physiology. Renal physiology. 288(6)
We studied the signal pathway through which troglitazone (TRO) acts in inducing cellular acidosis in LLC-PK1-F+cells in relation to ammoniagenesis and DNA synthesis. Cells were grown to confluent monolayers in 30-mm chambers and monitored for intrace
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::631860e222c52f66caa34e299abf52b1
https://pubmed.ncbi.nlm.nih.gov/15687249
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