Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Robert Daniel Adam"'
Autor:
Andreea Sorina Afana, Laura Vasiliu, Radu Sascău, Robert Daniel Adam, Cristina Rădulescu, Sebastian Onciul, Eliza Cinteză, Adela Chirita-Emandi, Ruxandra Jurcuț
Publikováno v:
Journal of Cardiovascular Development and Disease, Vol 11, Iss 2, p 41 (2024)
Hypertrophic cardiomyopathy (HCM) is a heterogeneous genetic disorder, most often caused by sarcomeric gene mutations, with a small proportion due to variants in non-sarcomeric loci. Phospholamban (PLN) is a phosphoprotein associated with the cardiac
Externí odkaz:
https://doaj.org/article/829d05c4bd95452eb96d714d3fc7674b
Autor:
Robert Daniel Adam, Daniel Coriu, Andreea Jercan, Sorina Bădeliţă, Bogdan A. Popescu, Thibaud Damy, Ruxandra Jurcuţ
Publikováno v:
ESC Heart Failure, Vol 8, Iss 4, Pp 2380-2396 (2021)
Abstract Cardiac amyloidosis is a restrictive cardiomyopathy determined by the accumulation of amyloid, which is represented by misfolded protein fragments in the cardiac extracellular space. The main classification of systemic amyloidosis is determi
Externí odkaz:
https://doaj.org/article/c781c9da04a2447cb4974a9852f8cd07
Publikováno v:
Diagnostics, Vol 12, Iss 2, p 556 (2022)
Amyloidosis is a heterogeneous group of diseases caused by the extracellular deposition of amyloid insoluble fibrils in multiple organs, resulting in various clinical manifestations. Cardiac amyloidosis (CA) occurs mainly in primary light-chain (AL)
Externí odkaz:
https://doaj.org/article/2f32278a665f4b3f890c5b1328c18cee
Publikováno v:
Diagnostics; Volume 12; Issue 2; Pages: 556
Amyloidosis is a heterogeneous group of diseases caused by the extracellular deposition of amyloid insoluble fibrils in multiple organs, resulting in various clinical manifestations. Cardiac amyloidosis (CA) occurs mainly in primary light-chain (AL)