Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Rixin, Dai"'
Autor:
Qiang Su, Wanzhong Huang, Yuan Huang, Rixin Dai, Chen Chang, Qiu-Yan Li, Hao Liu, Zhenhao Li, Yuxiang Zhao, Qiang Wu, Di-Guang Pan
Publikováno v:
Cardiovascular Diabetology, Vol 23, Iss 1, Pp 1-16 (2024)
Abstract Background Diabetic cardiomyopathy (DCM) poses a growing health threat, elevating heart failure risk in diabetic individuals. Understanding DCM is crucial, with fibroblasts and endothelial cells playing pivotal roles in driving myocardial fi
Externí odkaz:
https://doaj.org/article/0cf49cf7d0d94849a87d87fdfe6a2389
Publikováno v:
Molecular Therapy: Nucleic Acids, Vol 35, Iss 1, Pp 102106- (2024)
Externí odkaz:
https://doaj.org/article/3b30ba6bd7bb444cb3bf73e910c2cedf
Publikováno v:
Scientific Reports, Vol 12, Iss 1, Pp 1-11 (2022)
Abstract MALAT1 was reported to sponge miR-30e, miR-126 and miR-155 in the pathogenesis of many diseases. Plasma miR-30e can indicate the risk of no-reflow during primary percutaneous coronary intervention (pPCI), while miR-126 can be used as a predi
Externí odkaz:
https://doaj.org/article/1c5a04f1fcfb419aa8f2e60f070a4db1
Publikováno v:
Molecular Therapy: Nucleic Acids, Vol 23, Iss , Pp 1258-1271 (2021)
Increasing evidence suggests that mitochondrial microRNAs (miRNAs) are implicated in the pathogenesis of cardiovascular diseases; however, their roles in ischemic heart disease remain unclear. Herein, we demonstrate that miR-146a is enriched in the m
Externí odkaz:
https://doaj.org/article/94c850735bec4203803b91794e48ced2
Autor:
Rixin Dai, Yanling Ren, Xiangwei Lv, Chen Chang, Shirong He, Quanzhong Li, Xiheng Yang, Lei Ren, Riming Wei, Qiang Su
Publikováno v:
American Journal of Physiology-Cell Physiology. 324:C222-C235
This study investigates the mechanism by which microRNA (miR)-30e-3p reduces coronary microembolism (CME)-induced cardiomyocyte pyroptosis and inflammation. Cardiac function tests, histological staining, and transmission electron microscopy were perf
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::46a3b8802a480619392b8fc147e1f5dd
https://doi.org/10.1152/ajpcell.00351.2022
https://doi.org/10.1152/ajpcell.00351.2022
Autor:
Yuli Xu, Xiangwei Lv, Ruping Cai, Yanling Ren, Shirong He, Wei Zhang, Quanzhong Li, Xiheng Yang, Rixin Dai, Riming Wei, Qiang Su
Publikováno v:
Journal of Molecular Medicine. 100:763-780
This study aims to explore the mechanism underlying miR-142-3p regulating myocardial injury induced by coronary microembolization (CME) through ATXN1L. miR-142-3p overexpression or ATXN1L knockout adenovirus vectors were injected into rats before CME
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c54263103971a29f824e4415c013b5e7
https://doi.org/10.1007/s00109-022-02198-z
https://doi.org/10.1007/s00109-022-02198-z
Autor:
Ruping, Cai, Yuli, Xu, Yanling, Ren, Shirong, He, Jing, Zheng, Binghui, Kong, Quanzhong, Li, Xiheng, Yang, Rixin, Dai, Riming, Wei, Qiang, Su
Publikováno v:
Apoptosis. 27:206-221
This study investigated how miR-136-5p partially affected cardiomyocyte pyroptosis in rats with coronary microembolization (CME). The cardiac function and structure of rats with CME were evaluated using echocardiography, hematoxylin and eosin stainin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1ecf7a986e201c29f324e66517269a6a
https://doi.org/10.1007/s10495-022-01712-5
https://doi.org/10.1007/s10495-022-01712-5
Publikováno v:
Korean Circulation Journal. 53:151
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2d975bdbe8e3b724a835d6e06f6e9aca
https://doi.org/10.4070/kcj.2022.0242
https://doi.org/10.4070/kcj.2022.0242
Publikováno v:
Scientific reports. 12(1)
MALAT1 was reported to sponge miR-30e, miR-126 and miR-155 in the pathogenesis of many diseases. Plasma miR-30e can indicate the risk of no-reflow during primary percutaneous coronary intervention (pPCI), while miR-126 can be used as a predictor of c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::37a307c4a91587b48b2840d235704119
https://pubmed.ncbi.nlm.nih.gov/35228564
https://pubmed.ncbi.nlm.nih.gov/35228564