Zobrazeno 1 - 10
of 75
pro vyhledávání: '"Rita Roberti"'
Autor:
Maya Petricciuolo, Magdalena Davidescu, Katia Fettucciari, Leonardo Gatticchi, Stefano Brancorsini, Rita Roberti, Lanfranco Corazzi, Lara Macchioni
Publikováno v:
Heliyon, Vol 6, Iss 12, Pp e05741- (2020)
Metabolic reprogramming of tumour cells sustains cancer progression. Similar to other cancer cells, glioblastoma cells exhibit an increased glycolytic flow, which encourages the use of antiglycolytics as an effective complementary therapy. We used th
Externí odkaz:
https://doaj.org/article/f46911debe074d51b6ac96eeedea2486
Autor:
Leonardo Gatticchi, Jose I. de las Heras, Aishwarya Sivakumar, Nikolaj Zuleger, Rita Roberti, Eric C. Schirmer
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 8 (2020)
Tissue-specific patterns of radial genome organization contribute to genome regulation and can be established by nuclear envelope proteins. Studies in this area often use cancer cell lines, and it is unclear how well such systems recapitulate genome
Externí odkaz:
https://doaj.org/article/6d453fadf9dc4d5a966ea2534975fcbc
Autor:
Leonardo Gatticchi, Ilaria Bellezza, Rachele Del Sordo, Matthew J Peirce, Angelo Sidoni, Rita Roberti, Alba Minelli
Publikováno v:
PLoS ONE, Vol 10, Iss 11, p e0141885 (2015)
Cholesterol is essential for diverse cellular functions and cellular and whole-body cholesterol homeostasis is highly controlled. Cholesterol can also influence cellular susceptibility to injury. The connection between cholesterol metabolism and infl
Externí odkaz:
https://doaj.org/article/cbf75f0446ef474c93a8bacc6bbcf2d9
Autor:
Veronica Ceccarelli, Giuseppe Nocentini, Monia Billi, Serena Racanicchi, Carlo Riccardi, Rita Roberti, Francesco Grignani, Luciano Binaglia, Alba Vecchini
Publikováno v:
PLoS ONE, Vol 9, Iss 1, p e85025 (2014)
Epigenetic alterations, including aberrant DNA methylation, contribute to tumor development and progression. Silencing of tumor suppressor genes may be ascribed to promoter DNA hypermethylation, a reversible phenomenon intensely investigated as poten
Externí odkaz:
https://doaj.org/article/5e8de31d6e8f42808583eb916c5bba87
Autor:
Ilaria Bellezza, Rita Roberti, Leonardo Gatticchi, Rachele Del Sordo, Maria Grazia Rambotti, Maria Cristina Marchetti, Angelo Sidoni, Alba Minelli
Publikováno v:
PLoS ONE, Vol 8, Iss 7, p e68017 (2013)
We have explored the role of Tm7sf2 gene, which codifies for 3β-hydroxysterol Δ14-reductase, an endoplasmic reticulum resident protein, in the sensitivity to endoplasmic reticulum stress and in the resulting inflammatory response. We used mouse emb
Externí odkaz:
https://doaj.org/article/2e1f03139f4d4796a67bb4a651ef18a7
Autor:
Nikolaj Zuleger, Jose I. de las Heras, Eric C. Schirmer, Aishwarya Sivakumar, Rita Roberti, Leonardo Gatticchi
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 8 (2020)
Gatticchi, L, de las Heras, J, Sivakumar, A, Zuleger, N, Roberti, R & Schirmer, E C 2020, ' Tm7sf2 disruption alters radial gene positioning in mouse liver leading to metabolic defects and diabetes characteristics ', Frontiers in Cell and Developmental Biology, vol. 8, 592573 . https://doi.org/10.3389/fcell.2020.592573
Frontiers in Cell and Developmental Biology
Gatticchi, L, de las Heras, J, Sivakumar, A, Zuleger, N, Roberti, R & Schirmer, E C 2020, ' Tm7sf2 disruption alters radial gene positioning in mouse liver leading to metabolic defects and diabetes characteristics ', Frontiers in Cell and Developmental Biology, vol. 8, 592573 . https://doi.org/10.3389/fcell.2020.592573
Frontiers in Cell and Developmental Biology
Tissue-specific patterns of radial genome organization contribute to genome regulation and can be established by nuclear envelope proteins. Studies in this area often use cancer cell lines, and it is unclear how well such systems recapitulate genome
Autor:
Leonardo Gatticchi, Lanfranco Corazzi, Magdalena Davidescu, Stefano Brancorsini, Lara Macchioni, Rita Roberti, Maya Petricciuolo, Katia Fettucciari
Publikováno v:
Heliyon
Heliyon, Vol 6, Iss 12, Pp e05741-(2020)
Heliyon, Vol 6, Iss 12, Pp e05741-(2020)
Metabolic reprogramming of tumour cells sustains cancer progression. Similar to other cancer cells, glioblastoma cells exhibit an increased glycolytic flow, which encourages the use of antiglycolytics as an effective complementary therapy. We used th
Autor:
Claudia Torino, Patrizia Pizzini, Sebastiano Cutrupi, Maurizio Postorino, Giovanni Tripepi, Francesca Mallamaci, Jochen Reiser, Carmine Zoccali, Giovanni Alati, Eleonora Barreca, Rosalia Boito, Margherita Bovino, Vincenzo Bruzzese, Maria Capria, Simonetta Cassani, Salvatore Chiarella, Antonio Chippari, Teresa Cicchetti, Edoardo Crifò-Gasparro, Carlo Curti, Francesco D’Agostino, Emanuela D’Anello, Maria De Gaudio, Aldo Foscaldi, Cesare Fornaciari, Corrado Franco, Alfredo Gaglioti, Domenico Galati, Francesco Grandinetti, Maurizio Gullo, Maria Rosa La Gamba, Domenico Logozzo, Iginia Maimone, Maria Letizia Mannino, Elena Mazzuca, Agazio Mellace, Giuseppe Natale, Vincenzo Panuccio, Domenico Plutino, Antonio Pugliese, Anna Reina, Rita Roberti, Mariagrazia Santangelo, Arcangelo Sellaro, Rosalba Scicchitano, Carmela Vardè, Francesco Zingone
Publikováno v:
Kidney International Reports
Introduction The soluble receptor of urokinase plasminogen activator (suPAR) is an innate immunity/inflammation biomarker predicting cardiovascular (CV) and non-CV events in various conditions, including type 2 diabetic patients on dialysis. However,
Autor:
Luigi Francesco Iannone, Vincenzo Micale, P. Tucci, Anella Saviano, C. De Caro, Emilio Russo, Valentina Nesci, Federica Raucci, Martina Tallarico, Antonio Leo, Marco Ferrari, S. Dimonte, Francesco Maione, Rita Roberti, M. M. Bove, A. L. Colia, Victorino Franco, S. Di Martino, Alessia Furgiuele, Marco Cosentino
Publikováno v:
Pharmadvances. 3:369
Autor:
Federica Susta, Lanfranco Corazzi, Luciano Binaglia, Maya Petricciuolo, Rita Roberti, Davide Chiasserini, Lara Macchioni, Emilia Castigli, Magdalena Davidescu, Pier Luigi Orvietani
Publikováno v:
Journal of Proteomics. 152:329-338
Glioblastoma (GBM) is the most common and aggressive brain tumour of adults. The metabolic phenotype of GBM cells is highly dependent on glycolysis; therefore, therapeutic strategies aimed at interfering with glycolytic pathways are under considerati