Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Risa Kamei"'
Autor:
Mai Thi Nhu Tran, Michito Hamada, Hyojung Jeon, Risako Shiraishi, Keigo Asano, Motochika Hattori, Megumi Nakamura, Yuki Imamura, Yuki Tsunakawa, Risa Fujii, Toshiaki Usui, Kaushalya Kulathunga, Christina-Sylvia Andrea, Ryusuke Koshida, Risa Kamei, Yurina Matsunaga, Makoto Kobayashi, Hisashi Oishi, Takashi Kudo, Satoru Takahashi
Publikováno v:
Nature Communications, Vol 8, Iss 1, Pp 1-14 (2017)
Complement component C1q activates efferocytosis, suppresses inflammatory responses, and is thereby thought to limit autoimmune disease. Here, the authors show that macrophage transcription factor MafB regulates total serum levels of C1q, which contr
Externí odkaz:
https://doaj.org/article/8ad6548fe15640be8a3fcc672a1b03f2
Publikováno v:
International Journal of Clinical Pharmacy
Background In recent years, rapid increase of elderly population has become a major social problem in developed countries. They tend to receive an increasing number of prescibed drugs due to multiple illnesses, which might include inappropriate medic
Autor:
Risa Kamei, Christina-Sylvia Andrea, Risa Fujii, Makoto Kobayashi, Motochika Hattori, Megumi Nakamura, Keigo Asano, Mai Thi Nhu Tran, Ryusuke Koshida, Yuki Imamura, Michito Hamada, Satoru Takahashi, Hyojung Jeon, Takashi Kudo, Hisashi Oishi, Kaushalya Kulathunga, Yurina Matsunaga, Toshiaki Usui, Yuki Tsunakawa, Risako Shiraishi
Publikováno v:
Nature Communications, Vol 8, Iss 1, Pp 1-14 (2017)
Nature Communications
Nature Communications
The transcription factor MafB is expressed by monocytes and macrophages. Efferocytosis (apoptotic cell uptake) by macrophages is important for inhibiting the development of autoimmune diseases, and is greatly reduced in Mafb-deficient macrophages. He
Autor:
Yuki Tsunakawa, Michito Hamada, Yuan‐Yu Lin, Satoru Takahashi, Kumiko Fujisawa, Hyojung Jeon, Kaushalya Kulathunga, Mai Thi Nhu Tran, Risa Kamei, Takashi Kudo, Megumi Nakamura
Publikováno v:
FEBS Open Bio
MafB, a transcription factor expressed selectively in macrophages, has important roles in some macrophage-related diseases, especially in atherosclerosis. In this study, we investigated the mechanism by which hematopoietic-specific MafB deficiency in
Autor:
Risa, Kamei, Toshifumi, Yamaoka, Kuniko, Ikinaga, Hiroyuki, Murota, Kentaro, Shimizu, Ichiro, Katayama
Publikováno v:
Clinical and experimental rheumatology. 35(4)