Zobrazeno 1 - 10
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pro vyhledávání: '"Richard M. Grocott-Mason"'
Publikováno v:
Endothelial Modulation of Cardiac Function ISBN: 9780367810559
Endothelial Modulation of Cardiac Function
Endothelial Modulation of Cardiac Function
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::601c84a1cd08be069b8605c325c5b813
https://doi.org/10.1201/9780367810559-2
https://doi.org/10.1201/9780367810559-2
Autor:
Richard M. Grocott-Mason, Ajay M. Shah
Publikováno v:
Intensive Care Medicine. 24:286-295
Autor:
Richard M. Grocott-Mason, Malcolm J. Lewis, Alexandre Mebazaa, Andrew H. Henderson, Walter J. Paulus, Ajay M. Shah, Chris B. Pepper
Publikováno v:
Progress in Cardiovascular Diseases. 39:263-284
Endothelial cells within the heart release a number of substances that modulate myocardial contractile function. These agents include nitric oxide, endothelin, prostanoids, adenylpurines, and other substances that have so far been characterized only
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 266:H1699-H1705
In isolated myocytes and papillary muscles, both nitric oxide, acting through guanosine 3',5'-cyclic monophosphate (cGMP), and cGMP analogues exert a novel effect on myocardial contraction, influencing mainly the onset of relaxation. We studied the e
Publikováno v:
Circulation. 101(2)
Background —Endothelin-1 (ET-1) is a potent positive inotrope in vitro, but its physiological effects on intrinsic myocardial contractile function in humans in vivo are unknown. Plasma ET-1 levels are elevated in heart failure, and ET-1 may be invo
Publikováno v:
Intensive care medicine. 24(9)
Publikováno v:
Endothelium : journal of endothelial cell research. 5(4)
ACE inhibitors exert both acute and chronic beneficial effects on cardiac function (e.g remodelling, diastolic dysfunction). We have previously reported that the ACE inhibitor captopril induces selective left ventricular (LV) relaxant effects in the
Publikováno v:
Circulation. 92(9)
Background ACE inhibitors exert both acute and chronic beneficial effects on cardiac function (eg, remodeling, diastolic dysfunction) in experimental studies and in patients. They inhibit the formation of angiotensin II as well as the degradation of
Publikováno v:
International journal of cardiology. 50(3)
Nitric oxide released by cardiac endothelial cells modulates myocardial contractile function through elevation of intracellular 3′,5′-cyclic guanosine monophosphate (cGMP). In the absence of agonist stimulation, nitric oxide typically enhances my
Publikováno v:
Scopus-Elsevier
Nitric oxide (NO) modulates myocardial contractile behavior in several isolated preparations, e.g., cardiac myocytes and papillary muscles, via elevation of intracellular guanosine 3',5'-cyclic monophosphate (cGMP). We have recently reported that the