Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Ricardo J. Restrepo"'
Autor:
Derek Wang, David A. Ford, William L. Neumann, Ronald J. Korthuis, Yajun Liu, Ricardo J. Restrepo, Hong Yu, Theodore Kalogeris, Meifang Wang
Publikováno v:
Am J Physiol Heart Circ Physiol
Myeloperoxidase (MPO)-derived hypochlorous (HOCl) reacts with membrane plasmalogens to yield α-chlorofatty aldehydes such as 2-chlorofatty aldehyde (2-ClFALD) and its metabolite 2-chlorofatty acid (2-ClFA). Recent studies showed that 2-ClFALD and 2-
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::591eb3bb9d99075aa1076d10c45e69f4
https://doi.org/10.1152/ajpheart.00440.2020
https://doi.org/10.1152/ajpheart.00440.2020
Autor:
Shivendra D. Shukla, Jacob D. Franke, Xuanyou Liu, Ronald J. Korthuis, Ricardo J. Restrepo, David A. Ford, Robert W. Lim, Annayya R. Aroor
Publikováno v:
J Pharmacol Exp Ther
Binge alcohol consumption is a health problem, but differences between the sexes remain poorly defined. We have examined the in vivo effects of three acute, repeat binge alcohol administration on the liver in male and female rats. Sprague-Dawley rats
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b41502570549f64cad3a91fd6d823532
https://europepmc.org/articles/PMC6695503/
https://europepmc.org/articles/PMC6695503/
Autor:
Kusum K. Kharbanda, Shivendra D. Shukla, Ricardo J. Restrepo, Jamal A. Ibdah, Annayya R. Aroor
Publikováno v:
Biomolecules; Volume 5; Issue 4; Pages: 3280-3294
Biomolecules
Biomolecules, Vol 5, Iss 4, Pp 3280-3294 (2015)
Biomolecules
Biomolecules, Vol 5, Iss 4, Pp 3280-3294 (2015)
Chronic alcoholics who also binge drink (i.e., acute on chronic) are prone to an exacerbated liver injury but its mechanism is not understood. We therefore investigated the in vivo effects of chronic and binge ethanol ingestion and compared to chroni
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c0c3cb711565718b97d3f19dcb295b16
https://doi.org/10.3390/biom5043280
https://doi.org/10.3390/biom5043280
Autor:
Peter Arvan, Bao Yan Zhang, Ricardo J. Restrepo, Harald Peter, Xiang Zhao, Steven F. Nothwehr
Publikováno v:
Traffic. 8:1841-1853
The penta-subunit retromer complex of yeast mediates selective retrieval of membrane proteins from the prevacuolar endosome to the trans Golgi network. In this study, we set out to generate a panel of vps35 dominant-negative mutants that disrupt retr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fa7c5bf4bd483b63a3c595c011979a1a
https://doi.org/10.1111/j.1600-0854.2007.00659.x
https://doi.org/10.1111/j.1600-0854.2007.00659.x
Publikováno v:
Hepatology international. 8
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::82c31dbc2520fe2e35dda4477a4854b9
https://pubmed.ncbi.nlm.nih.gov/26201320
https://pubmed.ncbi.nlm.nih.gov/26201320
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 354(1)
Ethanol and its major metabolite acetate both induced histone H3 acetylation in primary culture of rat hepatocytes. The acetylation by ethanol was dependent on the reactive oxygen species and mitogen-activated protein kinase pathway, whereas that by
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ead116f461e4749df49ab0831c725ceb
https://pubmed.ncbi.nlm.nih.gov/25886906
https://pubmed.ncbi.nlm.nih.gov/25886906
Publikováno v:
Hepatology international. 8
Ethanol binge augments liver injury after chronic ethanol consumption in humans, but the mechanism behind the enhanced liver injury by ethanol binge is not known. In this study we used a clinically relevant rat model in which liver injury is amplifie
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6af26fb4d0a76f0f8504c4ff1522b148
https://pubmed.ncbi.nlm.nih.gov/26201321
https://pubmed.ncbi.nlm.nih.gov/26201321
Publikováno v:
Proteome Science, Vol 10, Iss 1, p 29 (2012)
Proteome Science
Proteome Science
Background Binge ethanol in rats after chronic ethanol exposure augments necrosis and steatosis in the liver. In this study, two-dimensional gel electrophoresis proteomic profiles of liver of control, chronic ethanol, control-binge, and chronic ethan
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3c3b31d0074d6ceaf8a2c3b1773d3bf9
https://doaj.org/article/a3d99570e5a7445cbe8df5d422dbf9df
https://doaj.org/article/a3d99570e5a7445cbe8df5d422dbf9df