Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Ricardo A DeMoya"'
Publikováno v:
PLoS ONE, Vol 10, Iss 3, p e0119304 (2015)
MIR233 is genetically or epigenetically silenced in a subset of acute myeloid leukemia (AML). MIR223 is normally expressed throughout myeloid differentiation and highly expressed in hematopoietic stem cells (HSCs). However, the contribution of MIR223
Externí odkaz:
https://doaj.org/article/b6f2776026db483b93c3aba60efec152
Autor:
Ricardo A. DeMoya, Rachel E. Forman-Rubinsky, Deon Fontaine, Joseph Shin, Simon C. Watkins, Cecilia W. Lo, Michael Tsang
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 11 (2023)
Hypoplastic left heart syndrome (HLHS) is a congenital heart disease where the left ventricle is reduced in size. A forward genetic screen in mice identified SIN3A associated protein 130 kDa (Sap130), part of the chromatin modifying SIN3A/HDAC comple
Externí odkaz:
https://doaj.org/article/1e0da7b8f313498dbc762e7aba202c98
Autor:
Ricardo A. DeMoya, Rachel E. Forman-Rubinsky, Deon Fontaine, Joseph Shin, Simon C. Watkins, Cecilia Lo, Michael Tsang
Publikováno v:
bioRxiv
Hypoplastic left heart syndrome (HLHS) is a congenital heart disease where the left ventricle is reduced in size. A forward genetic screen in mice identified SIN3A associated protein 130kDa (Sap130), a protein in the chromatin modifying SIN3A/HDAC1 c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0ea3f314e3e655e6f5a888235c89e8d1
https://doi.org/10.1101/2023.03.30.534737
https://doi.org/10.1101/2023.03.30.534737
Publikováno v:
PLoS ONE, Vol 10, Iss 3, p e0119304 (2015)
PLoS ONE
PLoS ONE
MIR233 is genetically or epigenetically silenced in a subset of acute myeloid leukemia (AML). MIR223 is normally expressed throughout myeloid differentiation and highly expressed in hematopoietic stem cells (HSCs). However, the contribution of MIR223
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2f1812e50778ab255102f8dd3811e141
https://doi.org/10.1371/journal.pone.0119304
https://doi.org/10.1371/journal.pone.0119304