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pro vyhledávání: '"Rho Chi Huang"'
Supplementary data includes IC50 of SAHA and B390 (S1),bioinformatic analysis (S2 and S3), and tumor size (S4)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::41d43b71b29209385cbccf6f8ec4c219
https://doi.org/10.1158/1535-7163.22522542.v1
https://doi.org/10.1158/1535-7163.22522542.v1
Pancreatic ductal adenocarcinoma (PDAC) is a highly aggressive cancer characterized by early dissemination and poor drug response. Therefore, it is an unmet medical need to develop new strategies for treatment. As aberrant activation of ERK due to KR
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1030c6e663bf2a789641d9044ad53828
https://doi.org/10.1158/1535-7163.c.6543423.v1
https://doi.org/10.1158/1535-7163.c.6543423.v1
Publikováno v:
Molecular Cancer Therapeutics. 20:1550-1560
Pancreatic ductal adenocarcinoma (PDAC) is a highly aggressive cancer characterized by early dissemination and poor drug response. Therefore, it is an unmet medical need to develop new strategies for treatment. As aberrant activation of ERK due to KR
Background Pancreatic ductal adenocarcinoma (PDAC) is a highly aggressive cancer characterized by early dissemination and poor drug response. Loss-of-function of dual specificity phosphatase 2 (DUSP2), a critical regulator of MAPKs signaling, is high
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ec6e83d435ca546f71c874dc7346ea14
https://doi.org/10.21203/rs.3.rs-70831/v1
https://doi.org/10.21203/rs.3.rs-70831/v1