Zobrazeno 1 - 10
of 1 023
pro vyhledávání: '"Rh, Hruban"'
Autor:
Argani P, Christophe Rosty, Re, Reiter, Re, Wilentz, Sr, Murugesan, Sd, Leach, Ryu B, Hg, Skinner, Goggins M, Em, Jaffee, Cj, Yeo, Jl, Cameron, Se, Kern, Rh, Hruban
Publikováno v:
Europe PubMed Central
Serial analysis of gene expression (SAGE) can be used to quantify gene expression in human tissues. Comparison of gene expression levels in neoplastic tissues with those seen in nonneoplastic tissues can, in turn, identify novel tumor markers. Such m
Publikováno v:
Europe PubMed Central
Pancreatic adenocarcinoma is a major cause of cancer death, and yet little is known about its molecular pathogenesis. We identified p53 mutations in 19 (70%) of 27 primary pancreatic adenocarcinomas. Most were missense point mutations, and the mutati
Autor:
Ab, Seymour, Rh, Hruban, Redston M, Carlos Caldas, Sm, Powell, Kw, Kinzler, Cj, Yeo, Se, Kern
Publikováno v:
Europe PubMed Central
Knowledge of the patterns of allelic loss has been useful in identifying the spectrum of the tumor suppressor genes involved in various tumor types. Such analyses in pancreatic carcinoma have been difficult due to the characteristic host desmoplastic
Publikováno v:
Hepatology. 22:A145
Publikováno v:
Europe PubMed Central
During the early phase of a human cytomegalovirus (HCMV) infection, the 2.7-kb early gene is by far the most abundantly transcribed RNA. Using strand-specific 32P or digoxigenin-labeled riboprobes derived from a subgenomic fragment of the HCMV Towne
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::4626528538f313f3d75e75eefadcf8ee
http://europepmc.org/abstract/med/1332487
http://europepmc.org/abstract/med/1332487
Autor:
Simbolo M, Fassan M, Ruzzenente A, Mafficini A, Ld, Wood, Corbo V, Melisi D, Malleo G, Vicentini C, Malpeli G, Antonello D, Sperandio N, Capelli P, Tomezzoli A, Calogero IACONO, Rt, Lawlor, Bassi C, Rh, Hruban, Guglielmi A, Tortora G
Publikováno v:
Europe PubMed Central
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::1bac95b050cd25c488eb6f33aba449ca
http://europepmc.org/abstract/med/24867389
http://europepmc.org/abstract/med/24867389
Autor:
Tascilar M, Hg, Skinner, Christophe Rosty, Sohn T, Re, Wilentz, Gj, Offerhaus, Adsay V, Ra, Abrams, Jl, Cameron, Se, Kern, Cj, Yeo, Rh, Hruban, Goggins M
Publikováno v:
Europe PubMed Central
SMAD4 (also called Dpc4) is a tumor suppressor in the TGF-beta signaling pathway that is genetically inactivated in approximately 55% of all pancreatic adenocarcinomas. We investigated whether prognosis after surgical resection for invasive pancreati
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::5b274d99d917969e2e6c1c986d0233a8
http://europepmc.org/abstract/med/11751510
http://europepmc.org/abstract/med/11751510
Autor:
Tc, Wu, Rh, Hruban, Rf, Ambinder, Marie Pizzorno, Cameron DE, Wa, Baumgartner, Ba, Reitz, Gs, Hayward, Gm, Hutchins
Publikováno v:
Europe PubMed Central
Cytomegalovirus (CMV) infection has been associated with the development of accelerated arteriosclerosis in heart transplant recipients. This association prompted the authors to examine, using in situ hybridization techniques, the coronary arteries o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::b51cd48c15d256fb61e95b57a7cbd18f
http://europepmc.org/abstract/med/1312309
http://europepmc.org/abstract/med/1312309
Publikováno v:
Europe PubMed Central
Pancreatic adenocarcinoma is the fifth leading cause of cancer death in the United States. Mutations in the K-ras oncogene occur in 85% of pancreatic adenocarcinomas and have also been identified in 75% of pancreatic ducts with mucinous cell hyperpla
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::a113342a169517194a6dc7fcaf991f73
http://europepmc.org/abstract/med/8012983
http://europepmc.org/abstract/med/8012983
Autor:
Rh, Hruban, Pd, Sturm, Rj, Slebos, Re, Wilentz, Ar, Musler, Cj, Yeo, Ta, Sohn, Marie-Louise van Velthuysen, Gj, Offerhaus
Publikováno v:
Europe PubMed Central
It can be difficult to distinguish benign bile duct proliferations (BDPs) from well-differentiated metastatic peripancreatic adenocarcinomas on histological grounds alone. Most peripancreatic carcinomas harbor activating point mutations in codon 12 o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::2aa08ea8b63c622639b7a79bd9d0792a
http://europepmc.org/abstract/med/9327727
http://europepmc.org/abstract/med/9327727