Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Reyna L. VanGilder"'
Early activation of STAT3 regulates reactive astrogliosis induced by diverse forms of neurotoxicity.
Autor:
James P O'Callaghan, Kimberly A Kelly, Reyna L VanGilder, Michael V Sofroniew, Diane B Miller
Publikováno v:
PLoS ONE, Vol 9, Iss 7, p e102003 (2014)
Astrogliosis, a cellular response characterized by astrocytic hypertrophy and accumulation of GFAP, is a hallmark of all types of central nervous system (CNS) injuries. Potential signaling mechanisms driving the conversion of astrocytes into "reactiv
Externí odkaz:
https://doaj.org/article/cbfcb822450048b28e6137d6d12047c8
Autor:
Ryan C. Turner, Aric F. Logsdon, Charles L. Rosen, Zachary J. Naser, Jason D. Huber, Reyna L. VanGilder, Rae R. Matsumoto, Brandon Lucke-Wold
Publikováno v:
Neuroimmunology and neuroinflammation
AIM Over 7 million traumatic brain injuries (TBI) are reported each year in the United States. However, treatments and neuroprotection following TBI are limited because secondary injury cascades are poorly understood. Lipopolysaccharide (LPS) adminis
Autor:
Stephanie L. Rellick, Reyna L. VanGilder, Ann McBurney, Laurie A. Theeke, Jennifer A. Mallow, Julia Moore
Publikováno v:
International Journal of Nursing Sciences, Vol 3, Iss 3, Pp 242-251 (2016)
International journal of nursing sciences
International journal of nursing sciences
Objectives Loneliness is a biopsychosocial determinant of health and contributes to physical and psychological chronic illnesses, functional decline, and mortality in older adults. This paper presents the results of the first randomized trial of LIST
Autor:
Charles L. Rosen, Julian E. Bailes, Zachary J. Naser, Rae R. Matsumoto, Jason D. Huber, Reyna L. VanGilder, Brandon Lucke-Wold, Ryan C. Turner
Publikováno v:
Neurosurgery. 74:382-394
BACKGROUND:: Concussion remains a symptom-based diagnosis clinically, yet preclinical studies investigating traumatic brain injury, of which concussion is believed to represent a 'mild' form, emphasize histological endpoints with functional assessmen
Autor:
Todd J. Crocco, Taura L. Barr, Karen S. Wilson, Ryan C. Turner, Jason D. Huber, Laurie Gutmann, Stephen M. Davis, Reyna L. VanGilder, Eric Haney, Danielle M. Davidov, Paul D. Chantler, Charles L. Rosen, Laurie A. Theeke, Kyle R. Stinehart
Publikováno v:
Journal of Clinical Neuroscience. 21:547-553
C-reactive protein (CRP) is an inflammatory biomarker of inflammation and may reflect progression of vascular disease. Conflicting evidence suggests CRP may be a prognostic biomarker of ischemic stroke outcome. Most studies that have examined the rel
Publikováno v:
Brain Research Bulletin. 88:313-319
The molecular causality and response to stroke is complex. Yet, much of the literature examining the molecular response to stroke has focused on targeted pathways that have been well-characterized. Consequently, our understanding of stroke pathophysi
Publikováno v:
Experimental Brain Research. 197:23-34
Uncontrolled or poorly controlled blood glucose during diabetes is an important factor in worsened vascular function. While evidence suggests that hyperglycemia-induced oxidative stress plays a prominent role in development of microangiopathy of the
Publikováno v:
Stroke. 46
Introduction: Inflammatory signals released from ischemic brain tissue recruits peripheral immune cells to the brain following ischemic stroke (IS). The mechanisms behind this crosstalk between the brain and periphery are not well understood. Studies
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 291:H2660-H2668
This study investigated the effects of streptozotocin-induced diabetes on the functional integrity of the blood-brain barrier in the rat at 7, 28, 56, and 90 days, using vascular space markers ranging in size from 342 to 65,000 Da. We also examined t
Publikováno v:
Archives of Biochemistry and Biophysics. 431:207-214
Strains of Saccharomyces cerevisiae that express either the wild type or the amyotrophic lateral sclerosis-associated mutant human copper–zinc superoxide dismutase (SOD1) proteins A4V and G93A, respectively, in a yeast SOD1-deficient parent strain