Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Reegan Miller"'
Autor:
Chad Slawson, Jeffrey Thompson, Reegan Miller, Heather M. Wilkins, Russell H. Swerdlow, Sarah Griffith, Amy Qiang, Gentry K. Cork, Jarrod Flax
Publikováno v:
J Alzheimers Dis
Background: Alzheimer’s disease (AD) features reductions in key bioenergetic fluxes and perturbed mitochondrial function. Cytoplasmic hybrids (cybrids) generated through the transfer of AD subject mitochondria to mtDNA-depleted SH-SY5Y neuroblastom
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::653a4bba9ce609592e0b8588e0034000
https://doi.org/10.3233/jad-200996
https://doi.org/10.3233/jad-200996
Autor:
Susan Kovats, Anna Karlik, Sean Turner, Reegan Miller, Erola Ainsua-Enrich, Ibrahim Hatipoglu, Harini Bagavant, Jose Alberola-Ila, Richard Pelikan, Sapana Kadel
Publikováno v:
The Journal of Immunology. 208:113.20-113.20
Biological sex differences in morbidity upon influenza A virus (IAV) infection are linked to stronger interferon-centered immune responses in females, yet the regulatory role of sex hormone receptors in immune cell subsets is incompletely understood.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::514ba9e8b472b25c2f4b44e1159a49b9
https://doi.org/10.4049/jimmunol.208.supp.113.20
https://doi.org/10.4049/jimmunol.208.supp.113.20
Publikováno v:
The Journal of Immunology. 206:55.15-55.15
Lung-resident group 2 innate lymphoid cells (ILC2s) produce type 2 cytokines and facilitate tissue repair in response to alarmins such as IL-33 released during respiratory virus infection, but they also may be functionally suppressed by type 1 cytoki
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0fab6fb67726ed0b91bb1db6bea141c1
https://doi.org/10.4049/jimmunol.206.supp.55.15
https://doi.org/10.4049/jimmunol.206.supp.55.15
Publikováno v:
The Journal of Immunology. 204:85.4-85.4
Lung-resident group 2 innate lymphoid cells (ILC2s) produce type 2 cytokines and facilitate tissue repair in response to alarmins such as IL-33 released during respiratory virus infection, but they also may be functionally suppressed by type 1 cytoki
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::df32a10ea1723c93b4c9913bc2154f74
https://doi.org/10.4049/jimmunol.204.supp.85.4
https://doi.org/10.4049/jimmunol.204.supp.85.4