Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Receptors, Interleukin/metabolism"'
Autor:
Maithri Prasad Aspari, Henning Glerup, Claus Johansen, Anne Grethe Jurik, René Østgård, Thomas Levin Andersen, Malene Hvid, Tue Wenzel Kragstrup, Bent Deleuran
Publikováno v:
Andersen, T, Østgård, R D, Aspari, M P, Kragstrup, T W, Glerup, H, Jurik, A G, Johansen, C, Hvid, M & Deleuran, B W 2019, ' Anti-tumor necrosis factor treatment increases both the Th17 and Th22 T helper subsets in spondyloarthritis ', APMIS, vol. 127, no. 12, pp. 789-796 . https://doi.org/10.1111/apm.12994
The aim was to examine anti-tumor necrosis factor α (anti-TNFα) therapy influence changes on Th17 and Th22 cells in patients with spondyloarthritis (SpA), and its correlation with changes in clinical and magnetic resonance imaging (MRI) activity an
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6db5bb939c3f6525b159122b597a8a96
https://pure.au.dk/portal/da/publications/antitumor-necrosis-factor-treatment-increases-both-the-th17-and-th22-t-helper-subsets-in-spondyloarthritis(287d5617-ed7b-4d70-9955-d0957f458382).html
https://pure.au.dk/portal/da/publications/antitumor-necrosis-factor-treatment-increases-both-the-th17-and-th22-t-helper-subsets-in-spondyloarthritis(287d5617-ed7b-4d70-9955-d0957f458382).html
Publikováno v:
Cells
von Essen, M R, Søndergaard, H B, Petersen, E R S & Sellebjerg, F 2019, ' IL-6, IL-12, and IL-23 STAT-Pathway Genetic Risk and Responsiveness of Lymphocytes in Patients with Multiple Sclerosis ', Cells, vol. 8, no. 3, 285 . https://doi.org/10.3390/cells8030285
Cells, Vol 8, Iss 3, p 285 (2019)
Volume 8
Issue 3
von Essen, M R, Søndergaard, H B, Petersen, E R S & Sellebjerg, F 2019, ' IL-6, IL-12, and IL-23 STAT-Pathway Genetic Risk and Responsiveness of Lymphocytes in Patients with Multiple Sclerosis ', Cells, vol. 8, no. 3, 285 . https://doi.org/10.3390/cells8030285
Cells, Vol 8, Iss 3, p 285 (2019)
Volume 8
Issue 3
Multiple sclerosis (MS) is an immune-mediated demyelinating disease characterized by central nervous system (CNS) lymphocyte infiltration, abundant production of pro-inflammatory cytokines, and inappropriate activation of Th1 and Th17 cells, B cells,
Publikováno v:
Immunological Reviews, Vol. 281, No 1 (2018) pp. 169-178
The interleukin (IL)-36 cytokines include 3 agonists, IL-36α, IL-36β, and IL-36γ that bind to a common receptor composed of IL-36R and IL-1RAcP to stimulate inflammatory responses. IL-36Ra is a natural antagonist that binds to IL-36R, but does not
Autor:
Guillaume Perriard, Mathieu Canales, Melanie Gentner, Lukas Enz, Myriam Schluep, Nicole Schaeren-Wiemers, Renaud Du Pasquier, Amandine Mathias
Publikováno v:
Journal of Neuroinflammation
Journal of Neuroinflammation, vol. 12, pp. 119
Journal of Neuroinflammation, vol. 12, pp. 119
Background Increasing evidences link T helper 17 (Th17) cells with multiple sclerosis (MS). In this context, interleukin-22 (IL-22), a Th17-linked cytokine, has been implicated in blood brain barrier breakdown and lymphocyte infiltration. Furthermore
Autor:
Poulin, Lionel F, Habran, Claude, Stordeur, Patrick, Goldman, Michel, McKenzie, Andrew, Van Snick, Jacques, Renauld, Jean-Christophe, Braun, Michel Y
Publikováno v:
European cytokine network, 16 (3
We recently showed that interleukin-9 (IL-9), a Th2 cytokine, promotes IL-5-mediated rejection of allografts in mice. This observation led us to investigate the functional link between IL-9 and IL-5 production during alloreactive T cell responses in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______2101::e0388de6732689cc366819dab219c3d1
http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/163243
http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/163243