Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Rebecca Barbaresso"'
Publikováno v:
Gynecology and Obstetrics Clinical Medicine, Vol 4, Iss 3 (2024)
Externí odkaz:
https://doaj.org/article/c348c44eba5f46f5a408df2a9022b9f7
Autor:
Manuel Blandino-Rosano, Rebecca Barbaresso, Margarita Jimenez-Palomares, Nadejda Bozadjieva, Joao Pedro Werneck-de-Castro, Masayuki Hatanaka, Raghavendra G. Mirmira, Nahum Sonenberg, Ming Liu, Markus A. Rüegg, Michael N. Hall, Ernesto Bernal-Mizrachi
Publikováno v:
Nature Communications, Vol 8, Iss 1, Pp 1-15 (2017)
Deregulation of mTORC1 pathway has been associated with several human diseases including diabetes, neurodegeneration and cancer. Here Blandino-Rosanoet al. show that mTORC1 signalling controls insulin secretion and β-cell maintenance by regulation o
Externí odkaz:
https://doaj.org/article/effc74d97aa34c3f83d6c968e3443703
Publikováno v:
Journal of Pediatric and Adolescent Gynecology. 34:250
Background Microperforate and imperforate hymen are ostensibly separate diagnoses on a spectrum of hymenal anomalies. The incidence of imperforate hymen is 1 in 1000 births, while the rate for microperforate hymen is unknown, with few case reports (1
Autor:
Nahum Sonenberg, Akiko Yanagiya, Aaron Bender, Margarita Jimenez-Palomares, Manuel Blandino-Rosano, Ming Liu, Rebecca Barbaresso, Ernesto Bernal-Mizrachi, Joshua O. Scheys, Liangyou Rui
Publikováno v:
Diabetes
The mammalian target of rapamycin complex 1 (mTORC1) regulates several biological processes, although the key downstream mechanisms responsible for these effects are poorly defined. Using mice with deletion of eukaryotic translation initiation factor
Autor:
Joao Pedro Werneck-de-Castro, Ernesto Bernal-Mizrachi, Nadejda Bozadjieva, Nahum Sonenberg, Masayuki Hatanaka, Raghavendra G. Mirmira, Margarita Jimenez-Palomares, Rebecca Barbaresso, Markus A. Rüegg, Ming Liu, Michael N. Hall, Manuel Blandino-Rosano
Publikováno v:
Nature Communications, Vol 8, Iss 1, Pp 1-15 (2017)
Nature Communications
Nature Communications
Deregulation of mTOR complex 1 (mTORC1) signalling increases the risk for metabolic diseases, including type 2 diabetes. Here we show that β-cell-specific loss of mTORC1 causes diabetes and β-cell failure due to defects in proliferation, autophagy,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3f985747ab1b850db3dec677fea71610
https://edoc.unibas.ch/55701/
https://edoc.unibas.ch/55701/