Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Rebecca B. Chapin"'
Autor:
Sabita Roy, Rebecca B. Chapin, Linda J. Brady, Paul S. Brady, Richard Charboneau, Kelly Cain, Roderick A. Barke
Publikováno v:
Journal of Surgical Research. 59:460-467
The transcription factors C/EBP alpha and C/EBP beta belong to the leucine-zipper C/EBP (CCAAT/enhancer binding protein) family of DNA-binding proteins. C/EBP alpha and C/EBP beta are expressed in the liver and are implicated in the control of transc
Publikováno v:
The Journal of Nutrition. 124:2365-2375
To study the role of nuclear regulatory proteins in mediating dietary effects, hepatic CCAAT/enhancer binding protein (C/EBP), mRNA and transcription rate were measured for C/EBP-alpha and C/EBP-beta in nutritional states that profoundly alter energy
Autor:
Richard Charboneau, Rebecca B. Chapin, Roderick A. Barke, Sabita Roy, Sundaram Ramakrishnan, Kelly J. Cain
Publikováno v:
Cellular immunology. 179(1)
Chronic treatment of mice with morphine affects the proliferation, differentiation, and function of immune cells. In the present study, we investigated the mechanism by which morphine inhibits phytohemagglutinin (PHA)/interleukin-1 (IL-1)-induced thy
Autor:
Roderick A. Barke, Linda J. Brady, Rebecca B. Chapin, Sabita Roy, Scott Birklid, Paul S. Brady
Publikováno v:
The Journal of surgical research. 60(1)
Peritoneal sepsis results in downregulation of the gene that codes for the hepatic mitochondrial enzyme carnitine palmitoyltransferase (CPT). The inhibition of hepatic CPT transcription by sepsis is thought to be mediated, in part, by increased expre
Publikováno v:
Archives of surgery (Chicago, Ill. : 1960). 130(11)
Objective: To investigate the hypothesis that a central dopaminergic mechanism may regulate hepatic c-fosand c-jungene expression following peritoneal sepsis. Methods: First, dopamine or vehicle was instilled into a stereotaxically placed intracerebr
Publikováno v:
Archives of surgery (Chicago, Ill. : 1960). 129(12)
Objective: To test the hypothesis that thymic involution following peritoneal sepsis is secondary to thymocyte programmed cell death. Design: We investigated the temporal response of thymic weight and thymic DNA fragmentation following peritoneal sep