Zobrazeno 1 - 10
of 76
pro vyhledávání: '"Re-Wen Wu"'
Autor:
Cheng-Hsiu Lu, Wei-Shiung Lian, Re-Wen Wu, Yu-Han Lin, Chia-Hao Su, Chuan-Lin Chen, Ming-Hong Tai, Yu-Shan Chen, Shao-Yu Wang, Chao-Cheng Chen, Feng-Sheng Wang
Publikováno v:
APL Bioengineering, Vol 8, Iss 3, Pp 036110-036110-12 (2024)
Cartilage damage, a common cause of osteoarthritis, requires medical imaging for accurate diagnosis of pathological changes. However, current instruments can acquire limited imaging information due to sensitivity and resolution issues. Therefore, mul
Externí odkaz:
https://doaj.org/article/9351c98798494bf99179ce41616565cd
Publikováno v:
Antioxidants, Vol 13, Iss 4, p 470 (2024)
Imbalanced osteogenic cell-mediated bone gain and osteoclastic remodeling accelerates the development of osteoporosis, which is the leading risk factor of disability in the elderly. Harmonizing the metabolic actions of bone-making cells and bone reso
Externí odkaz:
https://doaj.org/article/39e1afc5e1604e79b9b585a646d6ebe5
Autor:
Wei-Shiung Lian, Re-Wen Wu, Jih-Yang Ko, Yu-Shan Chen, Shao-Yu Wang, Chun-Ping Yu, Holger Jahr, Feng-Sheng Wang
Publikováno v:
Cell Death and Disease, Vol 13, Iss 6, Pp 1-11 (2022)
Abstract Epigenome alteration in chondrocytes correlates with osteoarthritis (OA) development. H3K27me3 demethylase UTX regulates tissue homeostasis and deterioration, while its role was not yet studied in articulating joint tissue in situ. We now un
Externí odkaz:
https://doaj.org/article/6fb744a909a24bbd9777401b0e528866
Publikováno v:
Biomedicines, Vol 10, Iss 11, p 2942 (2022)
Neuropeptide Y (NPY)-Y1 receptor (Y1R) signaling is known to negatively affect bone anabolism. Our study aimed at investigating the impact of NPY-Y1R signaling in the pathogenesis of glucocorticoid-related osteonecrosis of the femoral head (ONFH). Fe
Externí odkaz:
https://doaj.org/article/d7458818568f48c48fe4e7b2065d9f0b
Publikováno v:
Life, Vol 12, Iss 10, p 1588 (2022)
Shoulder stiffness (SS) is a disease that is fibroblastic and inflammatory in nature. Leptin is an adipokine-mediating the fibroblastic and inflammatory processes of various diseases. Our study tried to investigate the role of leptin in SS pathogenes
Externí odkaz:
https://doaj.org/article/8bbb8e96eb8f44428cdab29171ea8159
Autor:
Wei-Shiung Lian, Feng-Sheng Wang, Yu-Shan Chen, Ming-Hsien Tsai, How-Ran Chao, Holger Jahr, Re-Wen Wu, Jih-Yang Ko
Publikováno v:
Biomedicines, Vol 10, Iss 4, p 860 (2022)
Osteoporosis and osteoarthritis account for the leading causes of musculoskeletal dysfunction in older adults. Senescent chondrocyte overburden, inflammation, oxidative stress, subcellular organelle dysfunction, and genomic instability are prominent
Externí odkaz:
https://doaj.org/article/6a9759b5f87e48a28cb280066cd033c0
Autor:
Chen-Yu Chen, Re-Wen Wu, Nai-Wen Tsai, Mel S. Lee, Wei-Che Lin, Mei-Chi Hsu, Chih-Cheng Huang, Yun-Ru Lai, Chia-Te Kung, Hung-Chen Wang, Yu-Jih Su, Chih-Min Su, Sheng-Yuan Hsiao, Ben-Chung Cheng, Yi-Fang Chiang, Cheng-Hsien Lu
Publikováno v:
Journal of Translational Medicine, Vol 16, Iss 1, Pp 1-9 (2018)
Abstract Background Acute ischemic stroke is a leading cause of mortality and long-term disability, and profiles of endothelial progenitor cells (EPCs) reflect the degree of endothelial impairment. This study tested the hypothesis that hyperbaric oxy
Externí odkaz:
https://doaj.org/article/be6099b9fd1147df8e1c374408be3c2e
Autor:
Wei-Shiung Lian, Re-Wen Wu, Yu-Shan Chen, Jih-Yang Ko, Shao-Yu Wang, Holger Jahr, Feng-Sheng Wang
Publikováno v:
Antioxidants, Vol 10, Iss 8, p 1248 (2021)
Senescent osteoblast overburden accelerates bone mass loss. Little is understood about microRNA control of oxidative stress and osteoblast senescence in osteoporosis. We revealed an association between microRNA-29a (miR-29a) loss, oxidative stress ma
Externí odkaz:
https://doaj.org/article/6290c4d88e404015bd2de1402e25e859
Publikováno v:
Antioxidants, Vol 10, Iss 9, p 1394 (2021)
Bone-forming cells build mineralized microstructure and couple with bone-resorbing cells, harmonizing bone mineral acquisition, and remodeling to maintain bone mass homeostasis. Mitochondrial glycolysis and oxidative phosphorylation pathways together
Externí odkaz:
https://doaj.org/article/8102b905c4ca4f15b907621e245ddc4b
Autor:
Alice Y.W. Chang, Faith C.H. Li, Chi-Wei Huang, Julie C.C. Wu, Kuang-Yu Dai, Chang-Han Chen, Shau-Hsuan Li, Chia-Hao Su, Re-Wen Wu
Publikováno v:
Neurobiology of Disease, Vol 71, Iss , Pp 292-304 (2014)
Pressor response after stroke commonly leads to early death or susceptibility to stroke recurrence, and detailed mechanisms are still lacking. We assessed the hypothesis that the renin–angiotensin system contributes to pressor response after stroke
Externí odkaz:
https://doaj.org/article/794b318ef6354162a288feb80f1bb58c