Zobrazeno 1 - 10
of 52
pro vyhledávání: '"Raul A. Dela Cadena"'
Publikováno v:
Journal of Osteoporosis, Vol 2013 (2013)
Increased life expectancy and the need for long-term antiretroviral therapy have brought new challenges to the clinical management of HIV-infected individuals. The prevalence of osteoporosis and fractures is increased in HIV-infected patients; thus o
Externí odkaz:
https://doaj.org/article/4d566ddea82849d89ba0c78c2aaa1755
Autor:
Katherine J. Elliott, Michael V. Autieri, Raul A. DeLa Cadena, William J. Foster, Satoru Eguchi, Khatuna Gabunia, Mitali Ray, Sheri E. Kelemen, Allison B. Herman, Ross N. England
Publikováno v:
Journal of Molecular and Cellular Cardiology. 105:38-48
The transformation of vascular smooth muscle cells [VSMC] into foam cells leading to increased plaque size and decreased stability is a key, yet understudied step in atherogenesis. We reported that Interleukin-19 (IL-19), a novel, anti-inflammatory c
Autor:
Raul A. DeLa Cadena, Satya P. Kunapuli, Mario C. Rico, James J. Rough, Fabiola Del Carpio-Cano, Fayez F. Safadi, Joanne M. Manns
Publikováno v:
Thrombosis Research. 129:801-806
Activated factor X (FXa) and thrombin can up-regulate gene expression of connective tissue growth factor (CTGF/CCN2) on fibroblasts. Since tissue factor (TF) is expressed on these cells, we hypothesized that they may assemble the prothrombinase compl
Autor:
Fayez F. Safadi, Raul A. DeLa Cadena, Oneida A. Arosarena, Fabiola Del Carpio-Cano, Emeka Nwodim, Mario C. Rico
Publikováno v:
Journal of Cellular Physiology. 226:2943-2952
Current osteoinductive protein therapy utilizes bolus administration of large doses of bone morphogenetic proteins (BMPs), which is costly, and may not replicate normal bone healing. The limited in vivo biologic activity of BMPs requires the investig
Autor:
Fabiola Del Carpio-Cano, Raul A. DeLa Cadena, James J. Rough, Mario C. Rico, Satya P. Kunapuli
Publikováno v:
Current Vascular Pharmacology. 999:1-6
Biologic therapy for rheumatoid arthritis (RA) targets specific molecules that mediate and sustain the clinical manifestations of this complex illness. Compared with the general population, patients with RA die prematurely, in part due to associated
Autor:
Audrey B. Uknis, Raul A. DeLa Cadena, Satya P. Kunapuli, Mario C. Rico, Jeffrey B. Driban, Joanne M. Manns
Publikováno v:
Translational Research. 152:95-98
Thrombospondin-1 (TSP1/THBS1) plays a major role in the pathophysiology of rheumatoid arthritis (RA); however, its interface with the cytokine network involved in RA has not been delineated. Correlations were performed between plasma levels of TSP1 a
Autor:
Irma Isordia-Salas, Robert W. Colman, Robin A. Pixley, Raul A. DeLa Cadena, Julian L. Castaneda, Albert Adam, Irma M. Sainz, Alexis Agelan, Bo Liu, R. Balfour Sartor
Publikováno v:
Arthritis & Rheumatism. 52:2549-2552
Objective To compare inflammatory peripheral arthritis in wild-type and high molecular weight kininogen (HK)–deficient rats, both on the genetically susceptible Lewis background. Methods By backcrossing Brown-Norway HK-deficient rats with Lewis rat
Publikováno v:
Thrombosis Research. 116:533-543
Introduction Thrombospondin 1 (TSP1) has the ability to bind to HL-60 cells and to reversibly inhibit human neutrophil elastase (HNE). Human factor V (FV) can be cleaved by HNE thereby providing FV with cofactor activity (FVa HNE ). Experiments were
Autor:
Irma M. Sainz, Audrey B. Uknis, Robert W. Colman, Albert Adam, Walter K. Long, Robin A. Pixley, Alexis Agelan, John P. Gaughan, Irma Isordia-Salas, Raul A. DeLa Cadena, Ricardo G. Espinola
Publikováno v:
The American Journal of Pathology. 165:969-976
We reported that high-molecular weight kininogen is proangiogenic by releasing bradykinin and that a monoclonal antibody to high-molecular weight kininogen, C11C1, blocked its binding to endothelial cells. We now test if this antibody can prevent art
Autor:
Raul A. DeLa Cadena, Robert W. Colman
Publikováno v:
Sepsis. 3:143-146