Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Rasha M. El Refai"'
Publikováno v:
Laboratory Medicine. 50:34-41
Single-nucleotide polymorphisms (SNPs) in microRNA-146a (miRNA-146a) can be associated with the development of immune-system dysfunctions.The aim of this work is to correlate SNPs of miRNA-146a and its target gene, IRAK1, with susceptibility, clinica
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::651357989f8f03e1bbe2d55f4b9a0198
https://doi.org/10.1093/labmed/lmy033
https://doi.org/10.1093/labmed/lmy033
Publikováno v:
Blood Coagulation & Fibrinolysis. 27:691-695
Protein Z has been reported to exert an important role in inhibiting coagulation. Polymorphisms in the protein Z gene (PROZ) may affect protein Z levels and thus play a role in thrombosis. This study aimed to investigate the prevalence and clinical s
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c64c2ef049dcb9bf8af539770fd49274
https://doi.org/10.1097/mbc.0000000000000483
https://doi.org/10.1097/mbc.0000000000000483
Publikováno v:
Clinical Rheumatology. 35:1515-1520
The Janus kinase/signal transducers and activators of transcription (JAK/STAT) pathway is one of a handful of pleiotropic cascades used to transduce a multitude of signals for development and homeostasis in humans. It is the principal signaling mecha
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e0cbc7039e985b36400dfc1beb57ae7a
https://doi.org/10.1007/s10067-016-3250-9
https://doi.org/10.1007/s10067-016-3250-9
Publikováno v:
The Egyptian Rheumatologist. 37:S1-S6
Aim of the work The aim of this study was to identify the relationship between disease activity in SLE Egyptian females and the presence, severity and pattern of major depression in these patients. Patients and methods The study sample included 100 f
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8471843ac1e3ba8ce2f705abc6caa840
https://doi.org/10.1016/j.ejr.2015.09.007
https://doi.org/10.1016/j.ejr.2015.09.007
Autor:
Fatma Talaat, Nehal Hamdy El Gobashy, Khaled El Hadidi, Walaa Abdrahman, Shada Ghoniem, Nesreen Sobhy, Mervat Eissa, Tahsin El Hadidi, Doaa Hassan, Rasha M. El Refai, Basma Medhat Ali, Maha Shaaban, Noha Abdelbaki, Lobna A. Maged, Angie Y Yousri
Publikováno v:
Rheumatology. 56
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9b6308a5a43ef7f1c9bf1b44150a0991
https://doi.org/10.1093/rheumatology/kex062.323
https://doi.org/10.1093/rheumatology/kex062.323
Cytotoxic T lymphocyte antigen-4 gene polymorphism in systemic lupus erythematosus Egyptian patients
Publikováno v:
Comparative Clinical Pathology. 24:41-45
The human cytotoxic T lymphocyte antigen-4 (CTLA-4) gene has been confirmed to be associated with several autoimmune diseases. In this study, we investigated the CTLA-4 gene polymorphism in systemic lupus erythematosus (SLE) Egyptian patients. A poly
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7c7310a802dceefdfcfae9e82cdfe500
https://doi.org/10.1007/s00580-013-1852-z
https://doi.org/10.1007/s00580-013-1852-z
Publikováno v:
Immunological investigations. 45(3)
Systemic lupus erythematosus (SLE) is an autoimmune disease, with multiple genetic and environmental factors involved in its etiology. The toll-like receptor 9 (TLR9) gene has been reported to have important roles in the development and progression o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8ef04a6f705c9125cfcaa9773f78f69f
https://pubmed.ncbi.nlm.nih.gov/27018624
https://pubmed.ncbi.nlm.nih.gov/27018624
Publikováno v:
Comparative Clinical Pathology. 23:357-360
The T-box21 (TBX21) gene encodes the transcription factor T-bet (T-box expressed in T cells), which influences naive T lymphocyte development and has been implicated in the pathogenesis of many diseases. We aimed to assess the implication of the TBX2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f5b43a3bf2ae398cc42ab3005615f110
https://doi.org/10.1007/s00580-012-1623-2
https://doi.org/10.1007/s00580-012-1623-2