Zobrazeno 1 - 10
of 35
pro vyhledávání: '"Ranieri, Antonella"'
Autor:
Ranieri, Antonella
Protein phosphatase 2A (PP2A) holoenzymes are present in most cell types, including cardiac myocytes, where they provide a large proportion of serine (Ser, S) and threonine (Thr, T) phosphatase activity. PP2A family members comprise a catalytic C sub
Externí odkaz:
http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.689181
Publikováno v:
In Journal of Molecular and Cellular Cardiology February 2018 115:20-31
Publikováno v:
In Journal of Pharmacological and Toxicological Methods May-June 2016 79:60-71
Autor:
Haruki, Murakami, Ranieri, Antonella
Publikováno v:
Il Giappone, 1999 Jan 01. 39, 201-223.
Externí odkaz:
https://www.jstor.org/stable/20753054
Autor:
Ranieri, Antonella
Publikováno v:
Il Giappone, 1999 Jan 01. 39, 175-199.
Externí odkaz:
https://www.jstor.org/stable/20753053
Autor:
Puhl, Sarah Lena, Weeks, Kate, Guran, Alican, Ranieri, Antonella, Boknik, Peter, Kirchhefer, Uwe, Müller, Frank Ulrich, Avkiran, Metin
Publikováno v:
Cardiovascular Research
Puhl, S L, Weeks, K, Guran, A, Ranieri, A, Boknik, P, Kirchhefer, U, Müller, F U & Avkiran, M 2019, ' Role of type 2A phosphatase regulatory subunit B56α in regulating cardiac responses to β-adrenergic stimulation in vivo ', Cardiovascular Research, vol. 115, no. 3, pp. 519-529 . https://doi.org/10.1093/cvr/cvy230, https://doi.org/10.1093/cvr/cvy230
Puhl, S L, Weeks, K, Guran, A, Ranieri, A, Boknik, P, Kirchhefer, U, Müller, F U & Avkiran, M 2019, ' Role of type 2A phosphatase regulatory subunit B56α in regulating cardiac responses to β-adrenergic stimulation in vivo ', Cardiovascular Research, vol. 115, no. 3, pp. 519-529 . https://doi.org/10.1093/cvr/cvy230, https://doi.org/10.1093/cvr/cvy230
Aims B56α is a protein phosphatase 2A (PP2A) regulatory subunit that is highly expressed in the heart. We previously reported that cardiomyocyte B56α localizes to myofilaments under resting conditions and translocates to the cytosol in response to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::8b0f17bedcec8a366719f981c060f9f6
http://europepmc.org/articles/PMC6383118
http://europepmc.org/articles/PMC6383118
Autor:
Wilder, Catherine D E, Pavlaki, Nikoleta, Dursun, Tutku, Gyimah, Paul, Caldwell-Dunn, Ellice, Ranieri, Antonella, Lewis, Hannah R, Curtis, Michael J
Publikováno v:
Wilder, C D E, Pavlaki, N, Dursun, T, Gyimah, P, Caldwell-Dunn, E, Ranieri, A, Lewis, H R & Curtis, M J 2018, ' Facilitation of ischaemia-induced ventricular fibrillation by catecholamines is mediated by β1 and β2 agonism in the rat heart in vitro ', British Journal of Pharmacology, vol. 175, no. 10, pp. 1669-1690 . https://doi.org/10.1111/bph.14176
BACKGROUND AND PURPOSEAntiarrhythmic β-blockers are used in patients at risk of myocardial ischaemia, but the survival benefit and mechanisms are unclear.We hypothesized that β-blockers do not prevent ventricular fibrillation (VF) but instead inhib
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______2761::51c4ed4b25909bdf3aa5496b724a3ba0
https://kclpure.kcl.ac.uk/en/publications/f9f49c5c-a577-4946-b625-012375654ddb
https://kclpure.kcl.ac.uk/en/publications/f9f49c5c-a577-4946-b625-012375654ddb
Akademický článek
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Autor:
Longman, Michael R.1, Ranieri, Antonella2, Avkiran, Metin2, Snabaitis, Andrew K.1,2 a.snabaitis@kingston.ac.uk
Publikováno v:
PLoS ONE. Jan2014, Vol. 9 Issue 1, p1-10. 10p.
Autor:
Weeks, Kate L., Ranieri, Antonella, Karaś, Agnieszka, Bernardo, Bianca C., ASHCROFT, Alexandra, Molenaar, Chris, McMullen, Julie R., Avkiran, Metin
Publikováno v:
Weeks, K L, Ranieri, A, Karaś, A, Bernardo, B C, ASHCROFT, A, Molenaar, C, McMullen, J R & Avkiran, M 2017, ' βAdrenergic stimulation induces histone deacetylase 5 (HDAC5) nuclear accumulation in cardiomyocytes by B55α-PP2A-mediated dephosphorylation ', Journal of the American Heart Association, vol. 6, no. 4, e004861, pp. 1-14 . https://doi.org/10.1161/JAHA.116.004861
Background--Class IIa histone deacetylase (HDAC) isoforms such as HDAC5 are critical signal-responsive repressors of maladaptive cardiomyocyte hypertrophy, through nuclear interactions with transcription factors including myocyte enhancer factor-2.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______2761::533f949038fd2f28dd82b482485ec130
https://kclpure.kcl.ac.uk/en/publications/fa53a80a-c4e0-4a95-8f6f-5e730c1af186
https://kclpure.kcl.ac.uk/en/publications/fa53a80a-c4e0-4a95-8f6f-5e730c1af186