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Autor:
Matthew B. Hawn, Normand Leblanc, Michael Wiwchar, Fiona Cunningham, Ramon Jose Ayon, Iain A. Greenwood, Maria L. Valencik, Abigail S. Forrest, Joydeep Aoun, Cherie A. Singer
Publikováno v:
Am J Physiol Cell Physiol
This study explored the mechanism by which Ca2+-activated Cl−channels (CaCCs) encoded by the Tmem16a gene are regulated by calmodulin-dependent protein kinase II (CaMKII) and protein phosphatases 1 (PP1) and 2A (PP2A). Ca2+-activated Cl−currents