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of 5
pro vyhledávání: '"Rameez Zaidi"'
Autor:
Shing Fai Chan, Xiayu Huang, Scott R. McKercher, Rameez Zaidi, Shu-ichi Okamoto, Nobuki Nakanishi, Stuart A. Lipton
Publikováno v:
Genomics Data, Vol 3, Iss C, Pp 24-27 (2015)
The myocyte enhancer factor 2 (MEF2) family of transcription factors is highly expressed in the brain and constitutes a key determinant of neuronal survival, differentiation, and synaptic plasticity. However, genome-wide transcriptional profiling of
Externí odkaz:
https://doaj.org/article/73e8eb511c4f452183a4c921aa1f0c04
Autor:
Nobuki Nakanishi, Stuart A. Lipton, Rameez Zaidi, Shu-ichi Okamoto, Shing Fai Chan, Xiayu Huang, Scott R. McKercher
Publikováno v:
Genomics Data, Vol 3, Iss C, Pp 24-27 (2015)
Genomics Data
Genomics Data
[Briefly describe the contents of the Data in Brief article. Tell the reader the repository and reference number for the data in the abstract to.] The myocyte enhancer factor 2 (MEF2) family of transcription factors is highly expressed in the brain,
Autor:
Scott R. McKercher, Nobuki Nakanishi, Mohd Waseem Akhtar, Stuart A. Lipton, Laurence M. Brill, Shing Fai Chan, Shu-ichi Okamoto, Rameez Zaidi, Samuel Sances
Publikováno v:
The Journal of Neuroscience. 34:4640-4653
Mutations in the ataxia telangiectasia mutated (ATM) gene, which encodes a kinase critical for the normal DNA damage response, cause the neurodegenerative disorder ataxia-telangiectasia (AT). The substrates of ATM in the brain are poorly understood.
Autor:
Emily A. Holland, John R. Yates, Rossella Russo, Stuart A. Lipton, Lujian Liao, Shu-ichi Okamoto, Alexey Eroshkin, Florian Haun, Rana Nikzad, Sugato Banerjee, Tomohiro Nakamura, Scott R. McKercher, Rameez Zaidi
Publikováno v:
Experimental Neurology. 236:298-306
HIV/gp120 transgenic mice manifest neuropathological features similar to HIV-associated neurocognitive disorders (HAND) in humans, including astrogliosis, microglia activation, and decreased neuronal synapses. Here, proteomic screening of synaptosome
Autor:
Dongdong Yao, Dagmar E. Ehrnhoefer, Stuart A. Lipton, Marcus Kaul, Arjay Clemente, Peng Xia, Mahmoud A. Pouladi, H-S Vincent Chen, Michael R. Hayden, Rona K. Graham, Dongxian Zhang, Rameez Zaidi, Gary Tong, Maria Talantova, Shu-ichi Okamoto
Publikováno v:
Nature medicine
Nature medicine, vol 15, iss 12
Nature medicine, vol 15, iss 12
Huntington's disease is caused by an expanded CAG repeat in the gene encoding huntingtin (HTT), resulting in loss of striatal and cortical neurons. Given that the gene product is widely expressed, it remains unclear why neurons are selectively target