Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Ramón Pérez-Nuñez"'
Autor:
Marianne Brenet, Samuel Martínez, Ramón Pérez-Nuñez, Leonardo A. Pérez, Pamela Contreras, Jorge Díaz, Ana María Avalos, Pascal Schneider, Andrew F. G. Quest, Lisette Leyton
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 8 (2021)
Cancer cell adhesion to the vascular endothelium is an important step in tumor metastasis. Thy-1 (CD90), a cell adhesion molecule expressed in activated endothelial cells, has been implicated in melanoma metastasis by binding to integrins present in
Externí odkaz:
https://doaj.org/article/7837a62594974388907d6aa8a21149c8
Autor:
Raúl Lagos-Cabré, Alvaro Alvarez, Milene Kong, Francesca Burgos-Bravo, Areli Cárdenas, Edgardo Rojas-Mancilla, Ramón Pérez-Nuñez, Rodrigo Herrera-Molina, Fabiola Rojas, Pascal Schneider, Mario Herrera-Marschitz, Andrew F. G. Quest, Brigitte van Zundert, Lisette Leyton
Publikováno v:
Journal of Neuroinflammation, Vol 14, Iss 1, Pp 1-13 (2017)
Abstract Background Neuroinflammation involves cytokine release, astrocyte reactivity and migration. Neuronal Thy-1 promotes DITNC1 astrocyte migration by engaging αVβ3 Integrin and Syndecan-4. Primary astrocytes express low levels of these recepto
Externí odkaz:
https://doaj.org/article/098ca1bbe8ac4c6987fb170d55625e4a
Publikováno v:
Neural Regeneration Research, Vol 20, Iss 4, Pp 1031-1041 (2025)
Astrocytes are the most abundant type of glial cell in the central nervous system. Upon injury and inflammation, astrocytes become reactive and undergo morphological and functional changes. Depending on their phenotypic classification as A1 or A2, re
Externí odkaz:
https://doaj.org/article/9126bf4aab634a41a2063a842291be7d
Autor:
Leonardo A. Pérez, Esteban Palacios, María Fernanda González, Ignacio Leyton-Rivera, Samuel Martínez-Meza, Ramón Pérez-Núñez, Emanuel Jeldes, Ana María Avalos, Jorge Díaz, Lisette Leyton
Publikováno v:
International Journal of Molecular Sciences, Vol 25, Iss 17, p 9454 (2024)
Astrogliosis is a process by which astrocytes, when exposed to inflammation, exhibit hypertrophy, motility, and elevated expression of reactivity markers such as Glial Fibrillar Acidic Protein, Vimentin, and Connexin43. Since 1999, our laboratory in
Externí odkaz:
https://doaj.org/article/80d14d71618147b4b806ceb30ef3acc7
Autor:
Milene Kong, Edgardo Rojas-Mancilla, Alvaro Alvarez, Fabiola Rojas, Rodrigo Herrera-Molina, Brigitte van Zundert, Andrew F. G. Quest, Lisette Leyton, Mario Herrera-Marschitz, Ramón Pérez-Nuñez, Pascal Schneider, Raúl Lagos-Cabré, Francesca Burgos-Bravo, Areli Cárdenas
Publikováno v:
Journal of neuroinflammation, vol. 14, no. 1, pp. 194
Journal of Neuroinflammation, Vol 14, Iss 1, Pp 1-13 (2017)
Journal of Neuroinflammation, Vol 14, Iss 1, Pp 1-13 (2017)
Neuroinflammation involves cytokine release, astrocyte reactivity and migration. Neuronal Thy-1 promotes DITNC1 astrocyte migration by engaging α V β 3 Integrin and Syndecan-4. Primary astrocytes express low levels of these receptors and are unresp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9168ac8be9fe7e25eaa7594f815db88a
https://doi.org/10.1186/s12974-017-0968-5
https://doi.org/10.1186/s12974-017-0968-5
Autor:
Raúl, Lagos-Cabré, Alvaro, Alvarez, Milene, Kong, Francesca, Burgos-Bravo, Areli, Cárdenas, Edgardo, Rojas-Mancilla, Ramón, Pérez-Nuñez, Rodrigo, Herrera-Molina, Fabiola, Rojas, Pascal, Schneider, Mario, Herrera-Marschitz, Andrew F G, Quest, Brigitte, van Zundert, Lisette, Leyton
Publikováno v:
Journal of Neuroinflammation
Background Neuroinflammation involves cytokine release, astrocyte reactivity and migration. Neuronal Thy-1 promotes DITNC1 astrocyte migration by engaging αVβ3 Integrin and Syndecan-4. Primary astrocytes express low levels of these receptors and ar
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::61afab8f59ce604b6ad04aeebc6f656c
https://pubmed.ncbi.nlm.nih.gov/28962574
https://pubmed.ncbi.nlm.nih.gov/28962574
Autor:
Ramón Pérez-Núñez, Alejandro Chamorro, María Fernanda González, Pamela Contreras, Rocío Artigas, Alejandro H. Corvalán, Brigitte van Zundert, Christopher Reyes, Pablo R. Moya, Ana María Avalos, Pascal Schneider, Andrew F. G. Quest, Lisette Leyton
Publikováno v:
Journal of Neuroinflammation, Vol 20, Iss 1, Pp 1-19 (2023)
Abstract Background In response to brain injury or inflammation, astrocytes undergo hypertrophy, proliferate, and migrate to the damaged zone. These changes, collectively known as "astrogliosis", initially protect the brain; however, astrogliosis can
Externí odkaz:
https://doaj.org/article/8f88a252b77145c0982c107e62d40b7f
Autor:
Arlek Gonzalez-Jamett, Ramón Pérez-Nuñez, Ana M. Cárdenas, Jean-Vianney Barnier, Natalia Barraza, Pablo Caviedes
Publikováno v:
Neurotoxicity Research
Neurotoxicity Research, Springer Verlag, 2016, 30 (1), pp.76-87. ⟨10.1007/s12640-016-9613-9⟩
Neurotoxicity Research, Springer Verlag, 2016, 30 (1), pp.76-87. ⟨10.1007/s12640-016-9613-9⟩
International audience; In humans, Down syndrome (DS) is caused by the presence of an extra copy of autosome 21. The most striking finding in DS patients is intellectual disability and the onset of Alzheimer's disease (AD)-like neuropathology in adul
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ef6b4abb862bb7767d4e2869aed11ca4
https://hal.archives-ouvertes.fr/hal-01346998
https://hal.archives-ouvertes.fr/hal-01346998
Autor:
Christian Arriagada, Raúl Caviedes, Mario A. Acuña, Jorge Noriega, Ricardo Delgado, Ana M. Cárdenas, Juan Bacigalupo, Pablo Caviedes, Juan Segura-Aguilar, Ramón Pérez-Nuñez
Publikováno v:
NEUROTOXICITY RESEARCH
Artículos CONICYT
CONICYT Chile
instacron:CONICYT
Artículos CONICYT
CONICYT Chile
instacron:CONICYT
Human Down syndrome (DS) is determined by the trisomy of autosome 21 and is expressed by multiple abnormalities, being mental retardation the most striking feature. The condition results in altered electrical membrane properties (EMPs) of fetal neuro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::48baa446fb0c4dacdccee87fd9218603