Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Ralf D. Hess"'
Publikováno v:
Emerging Infectious Diseases, Vol 5, Iss 5, Pp 730-733 (1999)
Externí odkaz:
https://doaj.org/article/2fc25fa7922a42bb80cf0330bcbd4293
Publikováno v:
Journal of Clinical Virology
Publikováno v:
Journal of Clinical Virology
Publikováno v:
Journal of Clinical Virology
Autor:
Ralf D. Hess
Publikováno v:
Journal of Clinical Microbiology. 42:3381-3387
In 1968 Epstein-Barr virus (EBV; now human herpesvirus 4) was found to be the major cause of infectious mononucleosis (IM), a usually self-limited clinical syndrome ([10][1]). Only about 5% of adults in Western societies remain EBV uninfected; thus,
Publikováno v:
Emerging Infectious Diseases, Vol 5, Iss 5, Pp 730-733 (1999)
Autor:
Hans E. Schaefer, Andreas M. Zeiher, Christian Ihling, Ralf D. Hess, Judith Haendeler, Gustav Fraedrich, Grit Menzel
Publikováno v:
The Journal of Pathology. 185:303-312
Atherosclerosis is a fibroproliferative disease of the arterial intima. It was recently found that wild-type p53 (wt p53) accumulates in human atherosclerotic tissue. Wt p53 is a cell cycle regulator involved in DNA repair, DNA synthesis, cell differ
Autor:
Thomas Bauknecht, Edgar Wagner, Ralf D. Hess, Ulrich Falken, Thomas Brandstetter, Elena Ninci
Publikováno v:
International Journal of Cancer. 75:847-854
The ovarian adenocarcinoma cell line HEY was used as an in vitro model to study the influence of recombinant human granulocyte colony-stimulating factor (rhG-CSF) on epithelial tumours such as ovarian cancer. Serum-starved cells were treated with rhG
Autor:
Gerhard Brandner, Ralf D. Hess
Publikováno v:
Oncogene. 15:2501-2504
The biological state of the tumour suppressor proteins Rb and p53 is altered in papillomavirus- and SV40-transformed cells, due to interaction with the DNA tumour virus oncogene proteins E6/E7 and the tumour (T) antigen. Thus, the DNA damage response
Autor:
Ralf D. Hess, Susanne Paetzold, Gerhard Brandner, Markus Kuther, Dietmar G. Braun, C. Haessler
Publikováno v:
Cytometry. 20:81-85
The aim of this study was to quantitate the number of cell membrane-located SV40 large tumor antigen (large T) molecules of SV40-transformed cell lines by cytofluorimetric analysis. Five different SV40-transformed cell lines were labelled by either a