Zobrazeno 1 - 10
of 137
pro vyhledávání: '"Rainer B Zotz"'
Publikováno v:
Haematologica, Vol 108, Iss 10 (2023)
Externí odkaz:
https://doaj.org/article/17592bbf95a84633bb361f0331896519
Autor:
Giovanni Di Minno, Rainer B Zotz, Roseline d’Oiron, Niels Bindslev, Matteo Nicola Dario Di Minno, Man-Chiu Poon
Publikováno v:
Haematologica, Vol 100, Iss 8 (2015)
Standard treatment for Glanzmann thrombasthenia is platelet transfusion. Recombinant activated factor VII has been shown to be successful in patients with Glanzmann thrombasthenia with platelet antibodies or who are refractory to platelet transfusion
Externí odkaz:
https://doaj.org/article/88c377115db94f31a9346b963e18de20
Publikováno v:
Gynäkologische Endokrinologie. 20:263-271
Publikováno v:
TH Open, Vol 03, Iss 03, Pp e286-e294 (2019)
Background Standard treatment for Glanzmann thrombasthenia (GT), a severe inherited bleeding disorder, is platelet transfusion. Recombinant activated factor VII (rFVIIa) is reported to be effective in GT with platelet antibodies and/or refractoriness
Externí odkaz:
https://doaj.org/article/1a925aeab57a48c089e5574244aa1090
Autor:
Natalie Hellermann, Alexander Tischer, Cécile V. Denis, Reinhard Schneppenheim, Winfried März, Tobias Obser, Maria A. Brehm, Emma Ruoqi Xu, Volker Huck, Ulrike Klemm, Rainer B. Zotz, Matthew Auton, Matthias Wilmanns, Stefan W. Schneider
Publikováno v:
Blood. 133:356-365
The frequent von Willebrand factor (VWF) variant p.Phe2561Tyr is located within the C4 domain, which also harbors the platelet GPIIb/IIIa-binding RGD sequence. To investigate its potential effect on hemostasis, we genotyped 865 patients with coronary
Autor:
Stefan Rimbach, Sebastian Franik, Ludwig Wildt, Eckhard Schroll, Rainer B. Zotz, Tina Buchholz, Ronald Fischer, Anke Erath, Cornelia Jaursch-Hancke, Stefanie Förderreuther, Patricia G. Oppelt, Werner Harlfinger, Hans-Christoph Diener, Gert Naumann, Heiko B. G. Franz, Kai J. Bühling, Sabine Segerer, Ludwig Kiesel, Hannelore Rott, Joseph Neulen, Petra Stute, Ines Thonke, Bettina Toth, Clemens B. Tempfer, Günter Krämer, Viola Hach-Wunderle, K. König, Peymann Hadji, Jan Beyer-Westendorf, Claudia Schumann, Helga Seyler, Rupert Bauersachs, Jutta Pliefke
Publikováno v:
Geburtshilfe Frauenheilkd
Aims This is an official interdisciplinary guideline published and coordinated by the German Society for Gynecology and Obstetrics (DGGG), the Austrian Society for Gynecology and Obstetrics (OEGGG) and the Swiss Society for Gynecology and Obstetrics
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c880876a95bf27cb7b0fe60557fb493e
https://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&origin=inward&scp=85100769087
https://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&origin=inward&scp=85100769087
Autor:
Rupert Bauersachs, Hannelore Rott, Rainer B. Zotz, Birgit Linnemann, Christina Hart, Ute Scholz, F. Bergmann
Publikováno v:
Hamostaseologie. 40(5)
Venous thromboembolism (VTE) is a major cause of maternal morbidity during pregnancy and the postpartum period. Because there is a lack of adequate study data, management strategies for the prevention of VTE during pregnancy have mainly been deduced
Letter by Erbel and Zotz Regarding Article, 'North American Expert Review of Rotational Atherectomy'
Autor:
Rainer B. Zotz, Raimund Erbel
Publikováno v:
Circulation. Cardiovascular interventions. 12(7)
Publikováno v:
Gerhardt, A, Scharf, R E, Greer, I A & Zotz, R B 2016, ' Hereditary risk factors for thrombophilia and probability of venous thromboembolism during pregnancy and the puerperium ', Blood, vol. 128, no. 19, pp. 2343-2349 . https://doi.org/10.1182/blood-2016-03-703728
Venous thromboembolism (VTE) is a leading cause of maternal mortality. Few studies have evaluated the individual risk of gestational VTE associated with heritable thrombophilia, and current recommendations for antenatal thromboprophylaxis in women wi
Publikováno v:
Transfusion Medicine Reviews. 30:92-99
Glanzmann thrombasthenia (GT) is a rare inherited autosomal recessive bleeding disorder of platelet function caused by a quantitative or qualitative defect of platelet membrane glycoprotein IIb/IIIa (integrin αIIbβ3), a fibrinogen receptor required