Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Ragnhild V. Nome"'
Autor:
Erik Øie, Mona Høysæter Fenstad, Tor Henrik Anderson Tvedt, Erik Koldberg Amundsen, Ragnhild V. Nome
Publikováno v:
Tidsskrift for Den norske legeforening.
Autor:
Milada Cvancarova Småstuen, Cecilie E. Kiserud, Ragnhild V. Nome, Trine Bjøro, Sophie D. Fosså
Publikováno v:
Acta Oncologica
Purpose: Testicular cancer survivors (TCSs) have increased risk of reduced kidney function related to treatment burden, but longitudinal studies of renal outcome in aging TCSs have been lacking. This longitudinal study describes age- and treatment-re
Autor:
Trine Bjøro, Sophie D. Fosså, Bjørn Olav Åsvold, Ragnhild V. Nome, Cecilie E. Kiserud, Milada Cvancarova Småstuen
Publikováno v:
Acta Oncologica
Purpose Thyroid hypofunction is a late effect observed in several groups of cancer survivors, but has to date not been evaluated in-depth in testicular cancer survivors (TCSs). We investigated the prevalence of thyroid hypofunction in long-term TCSs
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::93f829c48d532960084e02d1f9c102d9
http://hdl.handle.net/10852/91510
http://hdl.handle.net/10852/91510
Autor:
Kristina H. Haugaa, Cecilie E. Kiserud, Thor Edvardsen, Ragnhild V. Nome, Hege Sagstuen Haugnes, A W Bjerring, Thomas M. Stokke, Sophie D. Fosså, Sebastian I. Sarvari
Aims Cisplatin-based chemotherapy (CBCT) is essential in the treatment of metastatic testicular cancer (TC) but has been associated with long-term risk of cardiovascular morbidity and mortality. Furthermore, cisplatin can be detected in the body deca
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::15dc7acb45739211c95ae91c37e66c3f
http://hdl.handle.net/10852/88078
http://hdl.handle.net/10852/88078
Autor:
Trine Bjøro, Johan Bjerner, Nils Bolstad, Sophie D. Fosså, Ragnhild V. Nome, Elisabeth Paus, Kjell Nustad, Rita Steen
Publikováno v:
Clinical biochemistry. 52
BACKGROUND Human Chorionic Gonadotropin (hCG) is produced by germ cell tumors, but can also be elevated in benign conditions such as primary hypogonadism, where hCG is produced by the pituitary gland. In our experience, the reference limits for hCG (
Publikováno v:
Scandinavian Journal of Urology. 49:211-217
The aim of this study was to assess treatment-related changes in prostate-specific antigen (PSA), total and bone alkaline phosphatase (total ALP, bone ALP), and changes on conventional bone scans in patients with metastatic castration-resistant prost
Autor:
Anne Hansen Ree, Gunhild Harman, Åse Bratland, Ragnhild V. Nome, Yvonne Andersson, Øystein Fodstad
Publikováno v:
Molecular Cancer Therapeutics. 4:1231-1238
In breast cancer, radiation has a central role in the treatment of brain metastasis, although tumor sensitivity might be limited. The tumor cell defense response to ionizing radiation involves activation of cell cycle checkpoint signaling. Histone de
Autor:
Yvonne Andersson, Trond Stokke, Anne Hansen Ree, Øystein Fodstad, Åse Bratland, Ragnhild V. Nome
Publikováno v:
Radiotherapy and Oncology. 72:305-310
Background and purpose The tumor cell defense response to ionizing radiation involves a temporary arrest at the cell cycle G 2 checkpoint, which is activated by a signaling cascade initiated by the ATM kinase response to DNA damage, ultimately leadin
Publikováno v:
Oncogene. 22:8952-8955
DNA damage activates the G2 cell cycle checkpoint to allow time for DNA repair before mitotic entry. The mechanism involves inhibition of the enzymatic activity for polo-like kinase 1 (Plk1), rendering Cdc25C with a basal phosphatase activity that is
Autor:
Anne Hansen, Ree, Trond, Stokke, Ase, Bratland, Sebastian, Patzke, Ragnhild V, Nome, Sigurd, Folkvord, Leonardo A, Meza-Zepeda, Kjersti, Flatmark, Oystein, Fodstad, Yvonne, Andersson
Publikováno v:
Anticancer research. 26(3A)
In order to determine temporal responses of cell cycle populations to DNA damage, a rational combination of cell cycle analyses is critical. Moreover, the targeting of cell cycle checkpoint responses may modify the cytotoxic effect of DNA damage.The