Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Radek, Blatny"'
Autor:
Jana Vcelakova, Radek Blatny, Zbynek Halbhuber, Michal Kolar, Ales Neuwirth, Lenka Petruzelkova, Tereza Ulmannova, Stanislava Kolouskova, Zdenek Sumnik, Pavlina Pithova, Maria Krivjanska, Dominik Filipp, Katerina Stechova
Publikováno v:
Journal of Diabetes Research, Vol 2013 (2013)
Type 1 Diabetes (T1D) is considered to be a T-helper- (Th-) 1 autoimmune disease; however, T1D pathogenesis likely involves many factors, and sufficient tools for autoreactive T cell detection for the study of this disease are currently lacking. In t
Externí odkaz:
https://doaj.org/article/4bd8b45aaf3f4dc0987a47f1c4b5e069
Autor:
Dominik Filipp, Katerina Stechova, Radek Blatny, Michal Kolar, Tereza Ulmannova, Maria Krivjanska, Lenka Petruzelkova, Zbynek Halbhuber, Stanislava Kolouskova, Pavlina Pithova, Zdenek Sumnik, Ales Neuwirth, Jana Vcelakova
Publikováno v:
Journal of Diabetes Research
Journal of Diabetes Research, Vol 2013 (2013)
Journal of Diabetes Research, Vol 2013 (2013)
Type 1 Diabetes (T1D) is considered to be a T-helper- (Th-) 1 autoimmune disease; however, T1D pathogenesis likely involves many factors, and sufficient tools for autoreactive T cell detection for the study of this disease are currently lacking. In t
Publikováno v:
Biology of Reproduction. 90
Autosomal trisomies and monosomies bring serious threats to embryonic development through transcriptional disarray caused primarily by the dosage effect of the aneuploid part of the genome. The present study compared the effect of a mouse-viable 30-M
Publikováno v:
Biology of reproduction. 90(6)
Autosomal trisomies and monosomies bring serious threats to embryonic development through transcriptional disarray caused primarily by the dosage effect of the aneuploid part of the genome. The present study compared the effect of a mouse-viable 30-M
Autor:
Olivier Thaunat, Emilie Groyer, Jamila Khallou-Laschet, Hannes Stockinger, Anh-Thu Gaston, Radek Blatny, Aditi Varthaman, Srini V. Kaveri, Hafid Ait-Oufella, Ziad Mallat, Antonino Nicoletti, Giuseppina Caligiuri
Publikováno v:
Journal of the American College of Cardiology. 50(4)
ObjectivesThis study was designed to evaluate whether replacing CD31 (PECAM-1) signaling can restore the regulation of lymphocyte activation and improve experimental atherosclerosis.BackgroundAtherosclerosis, the principal cause of myocardial infarct