IRF4 is required for migration of CD4 ⁺ T cells to the intestine but not for Th2 and Th17 cell maintenance.

Autor: Schmidt C; Department for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.; I. Department of Medicine, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Harberts A; Department for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.; I. Department of Medicine, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Reimers D; Department for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Bertram T; Department for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Voß LC; Department for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Schmid J; Department for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Lory NC; Department for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Spohn M; Clinic of Pediatric Hematology and Oncology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.; Research Institute Children's Cancer Center Hamburg, Hamburg, Germany.; Bioinformatics Core Unit, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Koch-Nolte F; Department for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Huber S; I. Department of Medicine, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Raczkowski F; Department for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Breloer M; Section for Molecular Biology and Immunology, Bernhard Nocht Institute for Tropical Medicine, Hamburg, Germany.; Department for Biology, University Hamburg, Hamburg, Germany., Mittrücker HW; Department for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.

Publikováno v: Frontiers in immunology [Front Immunol] 2023 Jul 03; Vol. 14, pp. 1182502. Date of Electronic Publication: 2023 Jul 03 (Print Publication: 2023).

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Deubiquitinating Enzyme UCH-L1 Promotes Dendritic Cell Antigen Cross-Presentation by Favoring Recycling of MHC Class I Molecules.

Autor: Reinicke AT; Institute for Cellular and Integrative Physiology, University Medical Center Hamburg-Eppendorf, 20246 Hamburg, Germany., Raczkowski F; Department of Immunology, University Medical Center Hamburg-Eppendorf, 20246 Hamburg, Germany; and., Mühlig M; Institute for Cellular and Integrative Physiology, University Medical Center Hamburg-Eppendorf, 20246 Hamburg, Germany., Schmucker P; Institute for Cellular and Integrative Physiology, University Medical Center Hamburg-Eppendorf, 20246 Hamburg, Germany., Lischke T; Department of Immunology, University Medical Center Hamburg-Eppendorf, 20246 Hamburg, Germany; and., Reichelt J; Institute for Cellular and Integrative Physiology, University Medical Center Hamburg-Eppendorf, 20246 Hamburg, Germany., Schneider E; Department of Immunology, University Medical Center Hamburg-Eppendorf, 20246 Hamburg, Germany; and., Zielinski S; Institute for Cellular and Integrative Physiology, University Medical Center Hamburg-Eppendorf, 20246 Hamburg, Germany., Sachs M; Institute for Cellular and Integrative Physiology, University Medical Center Hamburg-Eppendorf, 20246 Hamburg, Germany., Jurack E; Institute of Molecular Medicine and Experimental Immunology, University Clinic of Bonn, 53127 Bonn, Germany., Tolosa E; Department of Immunology, University Medical Center Hamburg-Eppendorf, 20246 Hamburg, Germany; and., Kurts C; Institute of Molecular Medicine and Experimental Immunology, University Clinic of Bonn, 53127 Bonn, Germany., Mittrücker HW; Department of Immunology, University Medical Center Hamburg-Eppendorf, 20246 Hamburg, Germany; and., Meyer-Schwesinger C; Institute for Cellular and Integrative Physiology, University Medical Center Hamburg-Eppendorf, 20246 Hamburg, Germany; c.meyer-schwesinger@uke.uni-hamburg.de.

Publikováno v: Journal of immunology (Baltimore, Md. : 1950) [J Immunol] 2019 Oct 01; Vol. 203 (7), pp. 1730-1742. Date of Electronic Publication: 2019 Sep 06.

CD39 is upregulated during activation of mouse and human T cells and attenuates the immune response to Listeria monocytogenes.

Autor: Raczkowski F; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Rissiek A; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Ricklefs I; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Heiss K; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Schumacher V; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Wundenberg K; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Haag F; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Koch-Nolte F; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Tolosa E; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany., Mittrücker HW; Institute for Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.

Publikováno v: PloS one [PLoS One] 2018 May 09; Vol. 13 (5), pp. e0197151. Date of Electronic Publication: 2018 May 09 (Print Publication: 2018).

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