Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Rachelle V. Morton"'
Autor:
Kelly M. Shepardson, Benjamin Schwarz, Kyle Larson, Rachelle V. Morton, John Avera, Kimberly McCoy, Alayna Caffrey, Ann Harmsen, Trevor Douglas, Agnieszka Rynda-Apple
Publikováno v:
mBio, Vol 15, Iss 8 (2024)
Externí odkaz:
https://doaj.org/article/c0d0e91da9d0443183322e9c08973f4a
Autor:
Kelly M. Shepardson, Benjamin Schwarz, Kyle Larson, Rachelle V. Morton, John Avera, Kimberly McCoy, Alayna Caffrey, Ann Harmsen, Trevor Douglas, Agnieszka Rynda-Apple
Publikováno v:
mBio, Vol 8, Iss 6 (2017)
ABSTRACT Although viruses and viral capsids induce rapid immune responses, little is known about viral pathogen-associated molecular patterns (PAMPs) that are exhibited on their surface. Here, we demonstrate that the repeating protein subunit pattern
Externí odkaz:
https://doaj.org/article/bf3aeed89b554e81bd3b7e859806801e
Autor:
Kelly M. Shepardson, Kyle Larson, Rachelle V. Morton, Justin R. Prigge, Edward E. Schmidt, Victor C. Huber, Agnieszka Rynda-Apple
Publikováno v:
mBio, Vol 7, Iss 3 (2016)
ABSTRACT Bacterial superinfections are a primary cause of death during influenza pandemics and epidemics. Type I interferon (IFN) signaling contributes to increased susceptibility of mice to bacterial superinfection around day 7 post-influenza A viru
Externí odkaz:
https://doaj.org/article/82538702d9634950a37811b419608ec6
Autor:
Anfin Erickson, Ann L. Harmsen, Laura E. Richert, Kyle Larson, Agnieszka Rynda-Apple, Allen G. Harmsen, Rachelle V. Morton
Publikováno v:
European Journal of Immunology. 44:3263-3272
Superinfection in mice at day 7 postinfluenza infection exacerbates bacterial pneumonia at least in part via downstream effects of increased IFN-γ signaling. Here we show that up to 3 days postinfluenza infection, mice have reduced susceptibility to
Autor:
Allen G. Harmsen, Agnieszka Rynda-Apple, Ann L. Harmsen, Soo Han, James Wiley, Rachelle V. Morton, Laura E. Richert, Trevor Douglas, Kyle Larson
Publikováno v:
European Journal of Immunology. 44:397-408
We show that the intranasal delivery of non-replicative virus-like particles (VLPs), which bear structural, but no antigenic similarities to respiratory pathogens, acted to prime the lungs of mice to facilitate heightened and accelerated primary immu
Autor:
Rachelle V. Morton, Agnieszka Rynda-Apple, Victor C. Huber, Justin R. Prigge, Kelly M. Shepardson, Edward E. Schmidt, Kyle Larson
Publikováno v:
mBio, Vol 7, Iss 3 (2016)
mBio
mBio, Vol 7, Iss 3, p e00506-16 (2016)
mBio
mBio, Vol 7, Iss 3, p e00506-16 (2016)
Bacterial superinfections are a primary cause of death during influenza pandemics and epidemics. Type I interferon (IFN) signaling contributes to increased susceptibility of mice to bacterial superinfection around day 7 post-influenza A virus (IAV) i