Zobrazeno 1 - 10
of 30
pro vyhledávání: '"Rachel L Kinsella"'
Autor:
Rachel L Kinsella, Jacqueline M Kimmey, Asya Smirnov, Reilly Woodson, Margaret R Gaggioli, Sthefany M Chavez, Darren Kreamalmeyer, Christina L Stallings
Publikováno v:
PLoS Biology, Vol 21, Iss 6, p e3002159 (2023)
The immune response to Mycobacterium tuberculosis infection determines tuberculosis disease outcomes, yet we have an incomplete understanding of what immune factors contribute to a protective immune response. Neutrophilic inflammation has been associ
Externí odkaz:
https://doaj.org/article/74e60a84f7e44c52a2d20d37607f9839
Publikováno v:
PLoS Pathogens, Vol 12, Iss 1, p e1005391 (2016)
Acinetobacter baumannii, A. nosocomialis, and A. pittii have recently emerged as opportunistic human pathogens capable of causing severe human disease; however, the molecular mechanisms employed by Acinetobacter to cause disease remain poorly underst
Externí odkaz:
https://doaj.org/article/c7e7ad7baf5b4caaa36425a1ecbf13a9
Autor:
Rachel L. Kinsella, Chanchal Sur Chowdhury, Asya Smirnov, Yassin Mreyoud, Jacqueline M. Kimmey, Ekaterina Esaulova, Darren Kreamalmeyer, Maxim N. Artyomov, Christina L. Stallings
SUMMARYInflammation is critical for controlling infections, but when left unchecked can cause tissue damage and disease. For tuberculosis, the leading cause of death due to infection1, host inflammation is responsible for the clinical symptoms2, morb
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0473fcfc8e1230667910377f692aa0a9
https://doi.org/10.1101/2023.03.18.533244
https://doi.org/10.1101/2023.03.18.533244
Autor:
Chanchal Sur Chowdhury, Rachel L. Kinsella, E. Michael Nehls, Sumanta K. Naik, Daniel S. Lane, Priyanka Talukdar, Sthefany M. Chavez, Asya Smirnov, Wandy Beatty, Darren Kreamalmeyer, Joshua T. Mattila, Christina L. Stallings
SUMMARYNeutrophils are the most abundant cell type in airways of tuberculosis patients. Recent investigations reported induction of neutrophil extracellular traps (NETs) duringMycobacterium tuberculosis(Mtb) infection, however, the molecular regulati
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8858024adb5b3bae9344260399837aad
https://doi.org/10.1101/2022.11.29.518376
https://doi.org/10.1101/2022.11.29.518376
Autor:
Siwei Feng, E. Michael Nehls, Rachel L. Kinsella, Sthefany M. Chavez, Sumanta K. Naik, Samuel R. McKee, Neha Dubey, Amanda Samuels, Amanda Swain, Xiaoyan Cui, Skyler V. Hendrix, Reilly Woodson, Darren Kreamalmeyer, Asya Smirnov, Maxim N. Artyomov, Herbert W. Virgin, Ya-Ting Wang, Christina L. Stallings
SummaryHost autophagy had been associated with the control ofMycobacterium tuberculosis(Mtb) infection due to its ability to sequesters microorganisms through a process termed “xenophagy”1–4. Xenophagy purportedly limits Mtb replication within
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::6d38c7b5b8c693590fe03ead5aa50b83
https://doi.org/10.1101/2022.11.04.515158
https://doi.org/10.1101/2022.11.04.515158
Autor:
Rachel L. Kinsella, Jacqueline M. Kimmey, Asya Smirnov, Reilly Woodson, Margaret R. Gaggioli, Sthefany M. Chavez, Darren Kreamalmeyer, Christina L. Stallings
The immune response toMycobacterium tuberculosisinfection determines tuberculosis disease outcomes, yet we have an incomplete understanding of what immune factors contribute to a protective immune response. Neutrophilic inflammation has been associat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::251849686a140ee25b424b2d6329efc5
https://doi.org/10.1101/2022.11.04.515221
https://doi.org/10.1101/2022.11.04.515221
Autor:
Chanchal Sur Chowdhury, Rachel L. Kinsella, E. Michael Nehls, Sumanta K. Naik, Daniel S. Lane, Priyanka Talukdar, Sthefany M. Chavez, Asya Smirnov, Wandy Beatty, Darren Kreamalmeyer, Joshua T. Mattila, Christina L. Stallings
Publikováno v:
SSRN Electronic Journal.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::13da22d44762d11b7e49306d19de9bf6
https://doi.org/10.2139/ssrn.4309034
https://doi.org/10.2139/ssrn.4309034
Publikováno v:
Immunity. 53:471-473
Summary Although programmed cell death can control intracellular bacterial replication, the role of cell death during systemic Salmonella infection remained elusive. In this issue of Immunity, Doerflinger et al. discover a critical but overlapping ro
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::65954379b32080ef15d042f268d1c6a2
https://doi.org/10.1016/j.immuni.2020.08.009
https://doi.org/10.1016/j.immuni.2020.08.009
Autor:
Rachel L. Kinsella, M. Florencia Haurat, Mario F. Feldman, Niraj H. Tolia, Darya Urusova, Nichole D. Salinas
Publikováno v:
Journal of Biological Chemistry. 294:13344-13354
Members of the Acinetobacter baumannii-calcoaceticus complex are nosocomial pathogens frequently causing multidrug-resistant infections that are increasing at alarming rates. A. baumannii has become the Gram-negative bacterium with the highest rate o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0e681a658c93afc3afbd718d3c2f7c72
https://doi.org/10.1074/jbc.ra119.009805
https://doi.org/10.1074/jbc.ra119.009805
Autor:
Martina Kulén, Christer Larsson, Fredrik Almqvist, K. Syam Krishnan, Erik Chorell, James A. D. Good, Mette R. Hansen, Gregory A. Harrison, Dennis X. Zhu, Jonathan Livny, Scott J. Hultgren, Kelly Flentie, Christina L. Stallings, Anders E. G. Lindgren, Leslie A. Weiss, Torbjörn Wixe, Hasan Tükenmez, Andrew G. Cairns, Christoffer Bengtsson, Souvik Sarkar, Samantha D. Solomon, Miranda E. Schene, Rachel L. Kinsella
Publikováno v:
Proceedings of the National Academy of Sciences. 116:10510-10517
Mycobacterium tuberculosis ( Mtb ) killed more people in 2017 than any other single infectious agent. This dangerous pathogen is able to withstand stresses imposed by the immune system and tolerate exposure to antibiotics, resulting in persistent inf
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0fc38cc536017cb4fd2b8f71cbd16357
https://doi.org/10.1073/pnas.1818009116
https://doi.org/10.1073/pnas.1818009116