Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Rachel K Stiner"'
Autor:
Nicholas Butowski, Jane Rabbitt, Mitchell P. Rosen, Jessica L. Chan, Joseph M. Letourneau, Jennifer Clarke, E.E. Niemasik, Rachel K Stiner, Nikita Sinha, Susan M. Chang, Michael D. Prados
Publikováno v:
Neurooncol Pract
Background No studies have examined the fertility priorities of women undergoing treatment for their glioma. Glioma patients frequently undergo chemotherapy as part of their treatment; however, it is unknown whether patients truly are aware of its po
Publikováno v:
4.1 Clinical Physiology, Exercise and Functional Imaging.
Background - Air trapping (AT) is a marker of obstructive lung diseases (OLD). However, its significance in the setting of normal FEV 1 /FVC is unclear. Methods - Lung function results from 646 patients who underwent PFT for OLD assessment between 20
Autor:
Brett M. Elicker, Mehrdad Arjomandi, Siyang Zeng, Paul D. Blanc, Warren M. Gold, Rachel K Stiner, Jason Keene, Ian D. van Koeverden, Bruce Bos, Jeroen Geerts
Publikováno v:
BMJ open respiratory research, vol 5, iss 1
BMJ Open Respiratory Research
BMJ Open Respiratory Research
IntroductionExposure to secondhand smoke (SHS) is associated with occult obstructive lung disease as evident by abnormal airflow indices representing small airway disease despite having preserved spirometry (normal forced expiratory volume in 1 s-to-
Inflammatory and Repair Pathways Induced in Human Bronchoalveolar Lavage Cells with Ozone Inhalation
Autor:
Andrea Tham, Mehrdad Arjomandi, Chun Chen, Rachel K Stiner, Pascale Leroy, Agnès Paquet, Hofer Wong, John R. Balmes, Rachel Tenney
Publikováno v:
PLoS ONE
PLoS ONE, Public Library of Science, 2015, 10 (6), pp.e0127283. ⟨10.1371/journal.pone.0127283⟩
PLoS ONE, Vol 10, Iss 6, p e0127283 (2015)
PLoS ONE, Public Library of Science, 2015, 10 (6), pp.e0127283. ⟨10.1371/journal.pone.0127283⟩
PLoS ONE, Vol 10, Iss 6, p e0127283 (2015)
Background Inhalation of ambient levels of ozone causes airway inflammation and epithelial injury. Methods To examine the responses of airway cells to ozone-induced oxidative injury, 19 subjects (7 with asthma) were exposed to clean air (0ppb), mediu