Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Rachel Hazan"'
Autor:
June Y Hou, Alicia Rodriguez-Gabin, Leleesha Samaraweera, Rachel Hazan, Gary L Goldberg, Susan Band Horwitz, Hayley M McDaid
Publikováno v:
PLoS ONE, Vol 11, Iss 3, p e0151750 (2016)
Externí odkaz:
https://doaj.org/article/191d07942caa49ad8b0328d48e8538e4
Autor:
June Y Hou, Alicia Rodriguez-Gabin, Leleesha Samaraweera, Rachel Hazan, Gary L Goldberg, Susan Band Horwitz, Hayley M McDaid
Publikováno v:
PLoS ONE, Vol 8, Iss 2, p e54103 (2013)
While the clinical benefit of MEK inhibitor (MEKi)-based therapy is well established in Raf mutant malignancies, its utility as a suppressor of hyperactive MAPK signaling in the absence of mutated Raf or Ras, is an area of ongoing research. MAPK acti
Externí odkaz:
https://doaj.org/article/e9e92161999f4cda9464fffe33e86bc2
Autor:
Adam Kotorashvili, Andrew Ramnauth, Christina Liu, Juan Lin, Kenny Ye, Ryung Kim, Rachel Hazan, Thomas Rohan, Susan Fineberg, Olivier Loudig
Publikováno v:
PLoS ONE, Vol 7, Iss 4, p e34683 (2012)
BACKGROUND: Retrospective studies of archived human specimens, with known clinical follow-up, are used to identify predictive and prognostic molecular markers of disease. Due to biochemical differences, however, formalin-fixed paraffin-embedded (FFPE
Externí odkaz:
https://doaj.org/article/5510c31fbb2f4b6981f0afe03dbc5ebe
Autor:
Huizhi Liang, Outhiriaradjou Benard, Kimita Suyama, Zuen Ren, Hao Wang, Larry Norton, Rachel Hazan
Publikováno v:
Cancer Research. 83:1228-1228
The epithelial to mesenchymal transition (EMT) is a dynamic process driven by carcinoma cells undergoing partial and/or complete EMT, leading to tumor heterogeneity and malignancy. Here we show that TCF1, a TCF7 gene product, promotes an epithelial/m
Autor:
Kei Kuroda, Raul Fleischmajer, Ronald E. Gordon, Rachel Hazan, Allen N. Sapadin, Yoshihiko Yamada, Noriyuki Iehara, Fernando Unda, Mark Lebwohl
Publikováno v:
Journal of Investigative Dermatology. 115:771-777
Psoriasis is most probably an inherited disease characterized by cell proliferation, angiogenesis, and an inflammatory process. The pathophysiology remains unknown, although an alteration in cell-cell and cell-matrix adhesion versus an autoimmune pro
Autor:
June Y, Hou, Alicia, Rodriguez-Gabin, Leleesha, Samaraweera, Leleesha, Samaweera, Rachel, Hazan, Gary L, Goldberg, Susan Band, Horwitz, Hayley M, McDaid
Publikováno v:
PLoS ONE
PLoS ONE, Vol 8, Iss 2, p e54103 (2013)
PLoS ONE, Vol 8, Iss 2, p e54103 (2013)
While the clinical benefit of MEK inhibitor (MEKi)-based therapy is well established in Raf mutant malignancies, its utility as a suppressor of hyperactive MAPK signaling in the absence of mutated Raf or Ras, is an area of ongoing research. MAPK acti
Autor:
Rachel Hazan
N-cadherin is expressed in highly invasive tumor cell lines which lack E-cadnerin expression. We transfected a weakly-metastatic and E-cadherin expressing breast cancer cell line, MCF-7, with N-cadherin and analyzed the effects on cell migration, inv
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4b66dda4e2acac742840661b0aa79e12
https://doi.org/10.21236/ada384147
https://doi.org/10.21236/ada384147