Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Racheal Wadlow"'
Autor:
Jilai Zhao, Jatin Patel, Simranpreet Kaur, Seen-Ling Sim, Ho Yi Wong, Cassandra Styke, Isabella Hogan, Sam Kahler, Hamish Hamilton, Racheal Wadlow, James Dight, Ghazaleh Hashemi, Laura Sormani, Edwige Roy, Mervin C. Yoder, Mathias Francois, Kiarash Khosrotehrani
Publikováno v:
Nature Communications, Vol 12, Iss 1, Pp 1-17 (2021)
How endothelial to mesenchymal transition is regulated in endovascular progenitors is unclear. Here, the authors show that blocking Sox9 expression in murine endovascular progenitors regulates this transition on skin wounding, affecting the size of s
Externí odkaz:
https://doaj.org/article/4916239cb44542b7875c762bd088d608
Autor:
Adriana C. Pliego Zamora, Hansini Ranasinghe, Jessica E. Lisle, Chun Ki Ng, Stephen Huang, Racheal Wadlow, Andrew M. Scott, Andrew W. Boyd, Christopher I. Slape
Publikováno v:
Cancers, Vol 13, Iss 15, p 3858 (2021)
We recently characterised the NUP98-HOXD13 (NHD13) mouse as a model of T-cell pre-leukaemia, featuring thymocytes that can engraft in recipient animals and progress to T-cell acute lymphoblastic leukaemia (T-ALL). However, loss of this engraftment ab
Externí odkaz:
https://doaj.org/article/51f29d90d7984faca589257edbacc472
Autor:
James Dight, Sam L. Kahler, Edwige Roy, Ho Yi Wong, Jatin Patel, Hamish Hamilton, Racheal Wadlow, Mathias Francois, Isabella Hogan, Seen Ling Sim, Simranpreet Kaur, Laura Sormani, Kiarash Khosrotehrani, Mervin C. Yoder, Jilai Zhao, Ghazaleh Hashemi, Cassandra Styke
Publikováno v:
Nature Communications, Vol 12, Iss 1, Pp 1-17 (2021)
Nature Communications
Nature Communications
Endothelial to mesenchymal transition (EndMT) is a leading cause of fibrosis and disease, however its mechanism has yet to be elucidated. The endothelium possesses a profound regenerative capacity to adapt and reorganize that is attributed to a popul
Publikováno v:
Experimental Hematology. 76:S85
EphA3, a receptor tyrosine kinase which mediates cell-cell interactions, is a potential therapeutic target for haematopoietic malignancies because of overexpression of EphA3 in patients with these diseases, along with negligible expression in healthy