Zobrazeno 1 - 10
of 21
pro vyhledávání: '"Racheal G. Akwii"'
Autor:
Behnaz Lahooti, Racheal G. Akwii, Dhavalkumar Patel, Siavash ShahbaziNia, Margarita Lamprou, Mahboubeh Madadi, Thomas J. Abbruscato, Aristotelis Astrinidis, Ulrich Bickel, Abraham Al-Ahmad, Nadezhda A. German, George Mattheolabakis, Constantinos M. Mikelis
Publikováno v:
Journal of Pharmacology and Experimental Therapeutics. 385:35-49
Targeting the Angiopoietin/Tie Pathway: Prospects for Treatment of Retinal and Respiratory Disorders
Publikováno v:
Drugs
Anti-angiogenic approaches have significantly advanced the treatment of vascular-related pathologies. The ephemeral outcome and known side effects of the current vascular endothelial growth factor (VEGF)-based anti-angiogenic treatments have intensif
Publikováno v:
Cells, Vol 8, Iss 5, p 471 (2019)
Angiopoietins 1−4 (Ang1−4) represent an important family of growth factors, whose activities are mediated through the tyrosine kinase receptors, Tie1 and Tie2. The best characterized are angiopoietin-1 (Ang1) and angiopoietin-2 (Ang2). Ang1 is a
Externí odkaz:
https://doaj.org/article/2f4c140c46504c0d8424e3e6aac9ef6a
Autor:
Constantinos M. Mikelis, Vasileios Oikonomou, Mihalis S. Lionakis, Laurence Wood, Fatema Tuz Zahra, Racheal G Akwii, Sanaullah Sajib, Paul Tullar
Publikováno v:
The FASEB Journal. 35
Autor:
Racheal G, Akwii, Md S, Sajib, Fatema T, Zahra, Hanumantha R, Madala, Kalkunte S, Srivenugopal, Constantinos M, Mikelis
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 2193
Lymphangiogenesis, the formation of lymphatic vessels from preexisting ones, is an important process in wound-healing physiology. Deregulation of lymphangiogenesis and lymphatic vascular remodeling have been implicated in a range of inflammatory cond
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 2193
Cutaneous wound healing is an intricate and multifaceted process. Despite these complexities, the distinct phases of wound healing provide a unique opportunity to evaluate the roles of different targets in these coordinated responses. This protocol d
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 2193
The small GTPase RhoA participates in actin and microtubule machinery, cell migration and invasion, gene expression, vesicular trafficking and cell cycle, and its dysregulation is a determining factor in many pathological conditions. Similar to other
Publikováno v:
Methods in Molecular Biology ISBN: 9781071608449
Cutaneous wound healing is an intricate and multifaceted process. Despite these complexities, the distinct phases of wound healing provide a unique opportunity to evaluate the roles of different targets in these coordinated responses. This protocol d
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::b87015ff9167add854d241fd7be1ad66
https://doi.org/10.1007/978-1-0716-0845-6_1
https://doi.org/10.1007/978-1-0716-0845-6_1
Autor:
Hanumantha Rao Madala, Fatema Tuz Zahra, Racheal G Akwii, Kalkunte S. Srivenugopal, Sanaullah Sajib, Constantinos M. Mikelis
Publikováno v:
Methods in Molecular Biology ISBN: 9781071608449
Lymphangiogenesis, the formation of lymphatic vessels from preexisting ones, is an important process in wound-healing physiology. Deregulation of lymphangiogenesis and lymphatic vascular remodeling have been implicated in a range of inflammatory cond
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::77a7d4f54955d848d421aa6df4268101
https://doi.org/10.1007/978-1-0716-0845-6_9
https://doi.org/10.1007/978-1-0716-0845-6_9
Publikováno v:
Methods in Molecular Biology ISBN: 9781071608449
The small GTPase RhoA participates in actin and microtubule machinery, cell migration and invasion, gene expression, vesicular trafficking and cell cycle, and its dysregulation is a determining factor in many pathological conditions. Similar to other
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9273a2b6b08dbd410f1650403c080d2d
https://doi.org/10.1007/978-1-0716-0845-6_10
https://doi.org/10.1007/978-1-0716-0845-6_10