Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Rachael J. Cerniway"'
Autor:
G. Paul Matherne, Marguerite M. Crawford, Amy R. Lankford, Rachael J. Cerniway, Sara E. Regan, Anne M. Byford
Publikováno v:
Drug Development Research. 58:439-446
Myocardial damage from ischemia/reperfusion injury can be minimized through endogenous protective mechanisms, including activation of A 1 adenosine receptors. Transgenic mice with cardiac-specific A 1 adenosine receptor overexpression (A 1 AR Trans)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a7581456807714a8959ceeefea5c65a5
https://doi.org/10.1002/ddr.10189
https://doi.org/10.1002/ddr.10189
Autor:
Amy R. Lankford, Anne M. Byford, Sara E. Regan, Marty W. Mayo, G. Paul Matherne, Rachael J. Cerniway, Michael Broad
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 284:H859-H866
We tested the hypothesis that myocardial ischemia-reperfusion (I/R)-induced apoptosis is attenuated in transgenic mice overexpressing cardiac A1 adenosine receptors. Isolated hearts from transgenic (TG, n = 19) and wild-type (WT, n = 22) mice underwe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9fe397c771f925bcc455198446b4ccc0
https://doi.org/10.1152/ajpheart.00251.2002
https://doi.org/10.1152/ajpheart.00251.2002
Autor:
Rachael J. Cerniway, Brent A. French, Stuart S. Berr, Zequan Yang, G. Paul Matherne, Anne M. Byford
Publikováno v:
American journal of physiology. Heart and circulatory physiology. 282(3)
Previous studies have shown that high-level (300-fold normal) cardiac overexpression of A1-adenosine receptors (A1-ARs) in transgenic (TG) mice protects isolated hearts against ischemia-reperfusion injury. However, this high level of overexpression i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5cbb0822cf606cd2848d283f576f8c4e
https://pubmed.ncbi.nlm.nih.gov/11834491
https://pubmed.ncbi.nlm.nih.gov/11834491
Autor:
G. Paul Matherne, Giovanni Ronca, Rachael J. Cerniway, R. Ray Morrison, Simonetta Ronca-Testoni, Riccardo Zucchi
Publikováno v:
Cardiovascular research. 53(2)
We investigated the effect of A(1) adenosine receptor overexpression, which has been reported to increase myocardial tolerance to ischemia-reperfusion injury, on sarcoplasmic reticulum (SR) Ca(2+) handling.Transgenic mouse hearts (approximately 300-f
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::986f06a1a4acbfbdd716d1618cfcdf31
https://pubmed.ncbi.nlm.nih.gov/11827676
https://pubmed.ncbi.nlm.nih.gov/11827676
Autor:
Rachael J. Cerniway, John P. Headrick, Kevin J. Ashton, Sara Regan, Stuart S. Berr, Jason Nigel John Peart, Glenn Harrison, G. Paul Matherne
Publikováno v:
Cardiovascular research. 53(1)
Objectives : To characterize effects of A3 adenosine receptor (A3AR) activation and gene knock-out on responses to ischemia-reperfusion in mouse heart. Methods : Perfused hearts from wild-type and A3AR gene knock-out (A3AR KO) mice were subjected to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b5356360ff710ef88c5eefa04c6b8d55
https://pubmed.ncbi.nlm.nih.gov/11744023
https://pubmed.ncbi.nlm.nih.gov/11744023
Publikováno v:
American journal of physiology. Heart and circulatory physiology. 281(4)
A3 adenosine receptors (A3ARs) have been implicated in regulating mast cell function and in cardioprotection during ischemia-reperfusion injury. The physiological role of A3ARs is unclear due to the lack of widely available selective antagonists. The
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b036d51d0d5957b84a52b3bc331208fc
https://pubmed.ncbi.nlm.nih.gov/11557567
https://pubmed.ncbi.nlm.nih.gov/11557567
Autor:
Amy R. Lankford, Rachael J. Cerniway, Sara E. Regan, Marguerite M. Crawford, Anne M. Byford, G. Paul Matherne
Publikováno v:
Drug Development Research; Apr2003, Vol. 58 Issue 4, p439-446, 8p