Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Raúl Flores-Vergara"'
Autor:
Magda C. Díaz-Vesga, Raúl Flores-Vergara, Jaime A. Riquelme, Marcelo Llancaqueo, Gina Sánchez, Cecilia Vergara, Luis Michea, Paulina Donoso, Andrew F. G. Quest, Ivonne Olmedo, Zully Pedrozo
Publikováno v:
International Journal of Molecular Sciences, Vol 24, Iss 1, p 667 (2022)
Cardiomyopathy is commonly observed in patients with autosomal dominant polycystic kidney disease (ADPKD), even when they have normal renal function and arterial pressure. The role of cardiomyocyte polycystin-1 (PC1) in cardiovascular pathophysiology
Externí odkaz:
https://doaj.org/article/7cd63e0bcbc3426fb8e4ebe377f29024
Autor:
Raúl Flores-Vergara, Ivonne Olmedo, Pablo Aránguiz, Jaime Andrés Riquelme, Raúl Vivar, Zully Pedrozo
Publikováno v:
Frontiers in Physiology, Vol 12 (2021)
Communication between cells is a foundational concept for understanding the physiology and pathology of biological systems. Paracrine/autocrine signaling, direct cell-to-cell interplay, and extracellular matrix interactions are three types of cell co
Externí odkaz:
https://doaj.org/article/f403dbe0f8c440d5b3178a1691f9e95a
Autor:
Carolina Fernández, Natalia Torrealba, Francisco Altamirano, Valeria Garrido-Moreno, César Vásquez-Trincado, Raúl Flores-Vergara, Camila López-Crisosto, María Paz Ocaranza, Mario Chiong, Zully Pedrozo, Sergio Lavandero
Publikováno v:
PLoS ONE, Vol 16, Iss 8, p e0255452 (2021)
Cardiac hypertrophy is the result of responses to various physiological or pathological stimuli. Recently, we showed that polycystin-1 participates in cardiomyocyte hypertrophy elicited by pressure overload and mechanical stress. Interestingly, polyc
Externí odkaz:
https://doaj.org/article/282852eddbe64e25877af76197d62e17
Publikováno v:
Circulation Research. 129
Background: Transverse tubules (T-tubules) play a key role in cardiac contractility. The expression of Bridging integrator 1 (BIN1), specifically, the cardiac isoforms BIN1+13 and BIN1+ 13+17 promote the formation and ultrastructure of T-tubules and
Autor:
Natalia Torrealba, Carolina Fernández, Camila López-Crisosto, César Vásquez-Trincado, Valeria Garrido-Moreno, Sergio Lavandero, Francisco Altamirano, María Paz Ocaranza, Raúl Flores-Vergara, Zully Pedrozo, Mario Chiong
Publikováno v:
PLoS ONE, Vol 16, Iss 8, p e0255452 (2021)
PLoS ONE
PLoS ONE
Cardiac hypertrophy is the result of responses to various physiological or pathological stimuli. Recently, we showed that polycystin-1 participates in cardiomyocyte hypertrophy elicited by pressure overload and mechanical stress. Interestingly, polyc