Zobrazeno 1 - 7
of 7
pro vyhledávání: '"R. Scott Heidersbach"'
Autor:
Shan L. Ward, Jeannette M. Asselin, Heidi R. Flori, R. Scott Heidersbach, Noelle Gorgis, Cynthia Fontana
Publikováno v:
Pediatr Emerg Care
Objective The aim of the study was to assess the association of initial lactate (L0) with mortality in children with severe sepsis. Methods This prospective cohort study included 74 patients younger than 18 years with severe sepsis admitted to the pe
Autor:
D. Michael McMullan, Jeffrey R. Fineman, Stephen M. Black, R. Scott Heidersbach, Michael J. Johengen, Janine M. Bekker
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 277:H1849-H1856
Life-threatening increases in pulmonary vascular resistance have been noted on acute withdrawal of inhaled nitric oxide (NO), although the mechanisms remain unknown. In vitro data suggest that exogenous NO exposure inhibits endothelial NO synthase (N
Publikováno v:
Pediatric Pulmonology. 28:3-11
Inhaled nitric oxide (NO) is currently used as an adjuvant therapy for a variety of pulmonary hypertensive disorders. In both animal and human studies, inhaled NO induces selective, dose-dependent pulmonary vasodilation. However, its potential intera
Autor:
D. Michael McMullan, Andrew J. Parry, R. Scott Heidersbach, Alexander A. Kon, Michael J. Johengen, Jeffrey R. Fineman, Stephen M. Black, Janine M. Bekker
Publikováno v:
Circulation. 102(19 Suppl 3)
After cardiopulmonary bypass (CPB), altered vascular reactivity is a major source of complications, particularly for children with increased pulmonary blood flow. Although changes in agonist-induced NO activity are well described after CPB, potential
Autor:
D. Michael McMullan, Janine M. Bekker, Andrew J. Parry, Michael J. Johengen, Alexander Kon, R. Scott Heidersbach, Stephen M. Black, Jeffrey R. Fineman
Publikováno v:
Circulation. 102
Background —After cardiopulmonary bypass (CPB), altered vascular reactivity is a major source of complications, particularly for children with increased pulmonary blood flow. Although changes in agonist-induced NO activity are well described after
Publikováno v:
Critical Care Medicine. 27:39A
Publikováno v:
Critical Care Medicine. 27:39A