Zobrazeno 1 - 4
of 4
pro vyhledávání: '"R. Queirό"'
Autor:
Santiago Muñoz-Fernández, A. Erra, Cesar Diaz-Torne, Paloma Vela, A Willisch-Domínguez, S. Marsal, Carlos González, Carlos Montilla, Hèctor Corominas, G Salvador, R. Queirό, Santos Castañeda, Raül Tortosa, Juan D. Cañete, J.J. Pérez-Venegas, Julio Ramirez, M. Lόpez-Lasanta, Jesús Tornero, Pedro Zarco, José Antonio Pinto, Adrià Aterido, J. Gratacόs, Simón Ángel Sánchez-Fernández, Juan Carlos Torre-Alonso, M. Lόpez-Corbeto, E. Rubio, Jesús Rodríguez, Ricardo Blanco, Daniel Roig, Antonio Fernández-Nebro, Antonio Julià, Andrea Pluma, Núria Palau, José Antonio Mosquera
Publikováno v:
Poster Presentations.
Background PsA has a higher heritability than PsV, indicating the existence of additional PsA-specific genetic factors. To date, however, the specific genetic basis underlying PsA is poorly understood. Objectives The objective the present study was t
Autor:
J. Cañete, J.A. Pinto, J. Gratacόs, R. Queirό, C. Ferrándiz, E. Fonseca, C. Montilla, J.C. Torre-Alonso, L. Puig, J.J. Pérez Venegas, A. Fernández Nebro, E. Fernández, S. Muñoz-Fernández, E. Daudén, C. González, D. Roig, J.L. Sánchez Carazo, P. Zarco, A. Erra, J.L. Lόpez Estebaranz, J. Rodríguez, D. Moreno Ramírez, P. de la Cueva, F. Vanaclocha, E. Herrera, S. Castañeda, E. Rubio, G. Salvador, C. Díaz-Torné, R. Blanco, A. Willisch Domínguez, J.A. Mosquera, P. Vela, J. Tornero, S. Sánchez-Fernández, H. Corominas, J. Ramírez, M. Lόpez-Lasanta, R. Tortosa, N. Palau, A. Alonso, A. Julià, S. Marsal
Publikováno v:
Annals of the Rheumatic Diseases. 74:190.2-190
Autor:
J.J. Pérez-Venegas, M. Lόpez-Lasanta, Antonio Julià, E. J. Herrera, Juan Carlos Torre-Alonso, Carlos Ferrándiz, Carlos González, J.L. Lόpez-Estebaranz, José Luis Fernández-Sueiro, Juan D. Cañete, Carlos Montilla, Eduardo Fonseca, A. Alonso, Raimon Sanmartí, Raül Tortosa, A. Erra, S. Marsal, Lluís Puig, I. Acosta, R. Queirό, P. Unamuno, Antonio Fernández-Nebro, Jesús Rodríguez, Francisco Vanaclocha, Pedro Zarco, J.L. Sánchez-Carazo, J. Gratacόs, David Moreno-Ramírez, Daniel Roig, J.M. Hernanz, Santiago Muñoz-Fernández, E. Daudén
Publikováno v:
Annals of the Rheumatic Diseases. 71:477.1-477
Background Psoriatic Arthritis (PsA) is a complex disease and is present in ∼11% in patients with Psoriasis (Ps). Recently, Genomewide Association Studies (GWAS) have expanded the number of risk loci for Ps in >20 new loci. Two of these genes, ERAP
Autor:
J.D. Cañete, J.M. Hernanz, E. Fonseca, C. Ferrandiz, P. Unamuno, L. Puig, J.L. Fernandez-Sueiro, R. Sanmartí, J. Rodriguez, J. Gratacόs, E. Dauden, J.L. Sánchez-Carazo, J.L. Lόpez-Estebaranz, D. Moreno, R. Queirό, J.C. Torre-Alonso, J.J. Pérez-Venegas, F. Vanaclocha, E. Herrera, S. Muñoz-Fernández, C. González, D. Roig, A. Erra, I. Acosta, A. Fernandez-Nebro, P. Zarco, A. Alonso, M. Lόpez-Lasanta, A. Julià, R. Tortosa, S. Marsal
Publikováno v:
Annals of the Rheumatic Diseases. 71:638.4-638
Background Psoriatic Arthritis (PsA) is a complex disease with a substantial genetic risk component (first-degree relative risk ∼55). Recently, Genomewide Association Studies (GWAS) have expanded the number of risk loci for Psoriasis (Ps) in >20 ne