Zobrazeno 1 - 10
of 34
pro vyhledávání: '"R. M. Raymond"'
Publikováno v:
Diabetes. 39:62-69
Autor:
R Y, Wang, R M, Raymond
Publikováno v:
Veterinary and human toxicology. 43(2)
To determine the site of thioridazine-induced cardiotoxicity and investigate the effectiveness of sodium bicarbonate (NaHCO3) therapy, isolated rat hearts were perfused with Krebs-Henseleit-Bicarbonate buffer (KHB) at a constant coronary flow of 10 m
Publikováno v:
Laboratory animal science. 45(6)
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 267(2)
Because of its positive inotropic effects that are independent of cyclic AMP, insulin was compared to epinephrine and glucagon as a novel treatment for cardiac toxicity from verapamil. Twenty-four alpha-chloralose-anesthetized mongrel canines of eith
Autor:
R M, Raymond
Publikováno v:
Circulatory shock. 30(1)
Early reports have attributed cardiac failure during acute and chronic models of shock to peripheral vascular dysfunction and decreased venous return. More recently interest has focused on the heart as a primary target responsible for cardiovascular
Autor:
R. M. Raymond, M. E. Ouinn
Publikováno v:
SHOCK. 1:33
Autor:
T. E. Emerson, R. M. Raymond
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 241:H134-H138
Theophylline, a competitive adenosine antagonist, was used to evaluate the role of adenosine in cerebral hypoxic hyperemia. Cerebral venous outflow was measured by the Rapela-Green technique in mongrel dogs anesthetized with pentobarbital sodium and
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 236:H600-H606
Bradykinin applied topically for 4 min produced marked dose-related increases in the number of fluorescent dextran (mol wt 145,000) vascular leakage sites exclusively from small postcapillary venules--evidence for an increase in macromolecular permea
Publikováno v:
Diabetes. 37:842-845
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 249:H715-H722
The slope of the left ventricular (LV) end-systolic pressure-diameter relationship (Ees) was analyzed in open-chest, pentobarbital-anesthetized dogs before and after endotoxin administration. A lead II electrocardiogram, systemic arterial pressure, L