Impaired activation of protein kinase C-zeta by insulin and phosphatidylinositol-3,4,5-(PO4)3 in cultured preadipocyte-derived adipocytes and myotubes of obese subjects

Autor: Atsushi Miura, Mini P. Sajan, Mary L. Standaert, R. Lea-Currie, Peter Vollenweider, D. M. Franklin, Gautam Bandyopadhyay, Robert V. Farese

Publikováno v: The Journal of clinical endocrinology and metabolism. 89(8)

Insulin resistance in obesity is partly due to diminished glucose transport in myocytes and adipocytes, but underlying mechanisms are uncertain. Insulin-stimulated glucose transport requires activation of phosphatidylinositol (PI) 3-kinase (3K), oper

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ccbb2df40bc5301bd10ef433b2f26198
https://pubmed.ncbi.nlm.nih.gov/15292339

Adipogenic potential of human adipose derived stromal cells from multiple donors is heterogeneous

Autor: A, Sen, Y R, Lea-Currie, D, Sujkowska, D M, Franklin, W O, Wilkison, Y D, Halvorsen, J M, Gimble

Publikováno v: Journal of cellular biochemistry. 81(2)

The current study was done to assess if heterogeneity existed in the degree of adipogenesis in stromal cells (preadipocytes) from multiple donors. In addition to conventional lipid-based methods, we have employed a novel signal amplification technolo

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::c94ac6ce297e3bb2fc0889e6001a072e
https://pubmed.ncbi.nlm.nih.gov/11241671

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PKC-zeta mediates insulin effects on glucose transport in cultured preadipocyte-derived human adipocytes.

Autor: Bandyopadhyay G; J. A. Haley Veterans' Hospital Research Service and Department of Internal Medicine, University of South Florida College of Medicine, Tampa, Florida 33612, USA. rfarese@com1.med.usf.edu, Sajan MP, Kanoh Y, Standaert ML, Quon MJ, Lea-Currie R, Sen A, Farese RV

Publikováno v: The Journal of clinical endocrinology and metabolism [J Clin Endocrinol Metab] 2002 Feb; Vol. 87 (2), pp. 716-23.