Zobrazeno 1 - 10
of 287
pro vyhledávání: '"R. Covenas"'
Publikováno v:
European Journal of Histochemistry, Vol 60, Iss 4 (2016)
Using an immunohistochemical technique, we have studied the distribution of 3-OH-anthranilic acid (3-HAA) in the rat brain. Our study was carried out in control animals and in rats in which a stroke model (single transient middle cerebral artery occl
Externí odkaz:
https://doaj.org/article/a32a6d05043d4b7fb7e77ec3c17eb0f4
Publikováno v:
European Journal of Histochemistry, Vol 60, Iss 3 (2016)
A highly specific monoclonal antibody directed against nitric oxide-tryptophan (NO-W) with good affinity (10-9 M) and specificity was developed. In the rat brain, using an indirect immunoperoxidase technique, cell bodies containing NO-W were exclusiv
Externí odkaz:
https://doaj.org/article/3f174eb261a643c3b6c8a19c3ac61f0e
Autor:
F. Werner, R. Covenas
Publikováno v:
Clinical Neurophysiology. 148:e20
Autor:
F.M. Werner, R. Covenas
Publikováno v:
Clinical Neurophysiology. 137:e21-e22
Autor:
F.M. Werner, R. Covenas
Publikováno v:
Clinical Neurophysiology. 131:e183-e184
Publikováno v:
Nucleus of the Solitary Tract ISBN: 9780429277214
This chapter provides some indications that inter alia angiotensin (ANG), neuropeptide Y, vasopressin, and cholecystokinin immunoreactive nerve terminal systems within the nucleus tractus solitaries may mainly operate via volume transmission (VT) and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4fa6a1404dc1c80398985710ea8de359
https://doi.org/10.1201/9780429277214-6
https://doi.org/10.1201/9780429277214-6
Autor:
F.M. Werner, R. Covenas
Publikováno v:
Clinical Neurophysiology. 129:e96-e97
Introduction In Alzheimer’s disease, neurotransmitter alterations, for example acetylcholine deficiency and a surplus of the excitotoxic glutamate have been described and are associated with cognitive impairment. An interaction between acetylcholin
Akademický článek
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Akademický článek
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Publikováno v:
Letters in Drug Design & Discovery. 3:138-148
Experimental Autoimmune Encephalomyelitis (EAE) was induced for the evaluation of a new drug candidate (GEM-SP) for multiple sclerosis. Using immunocytochemical techniques with a pan-leukocyte marker, "anti-CD 45", differential leukocyte infiltration